© Springer Nature Switzerland AG 2021
C. S. Foster et al. (eds.)Uveitishttps://doi.org/10.1007/978-3-030-52974-1_50

50. Other Vector-Borne Parasitic Infections

Koushik Tripathy1   and Aniruddha Agarwal2
(1)
Department of Vitreoretina and Uvea, ASG Eye Hospital, Kolkata, West Bengal, India
(2)
Department of Ophthalmology, Post Graduate Institute of Medical Education and Research (PGIMER), Advanced Eye Center, Chandigarh, India
 
Keywords
Vector-borne parasitic infectionsUveitisLeishmaniasisMalariaSeasonal hyperacute panuveitis

Leishmaniasis

Overview

  • Definition
    • Caused by protozoa (Leishmania species) which are transmitted by bites of infected sand flies (various species including Phlebotomus)

    • “Neglected tropical disease” mostly affecting poorest populations
      • Risk factors include malnutrition, weak immune system, poverty, poor housing, populations displacement, and deforestation

    • Presentation and management vary by species and affected population

    • Three forms of disease exist:
      • Cutaneous (most common) (CL)

      • Mucosal/mucocutaneous (Espundia) (ML)

      • Visceral (Kala-azar, black fever, most severe) (VL)

  • Symptoms
    • CL, ML – blurring, pain, redness, light sensitivity

    • VL – blurring, pain, redness, light sensitivity, floaters

  • Laterality
    • Typically unilateral

  • Course
    • Subacute onset with chronic self-limiting course
      • May have permanent sequelae in more severe disease

    • ML is often seen after untreated or unrecognized CL

  • Age of onset
    • All age groups affected

  • Gender/race
    • Any person in endemic area

  • Systemic association
    • CL – skin lesions mostly in exposed areas, may be diffuse

    • ML – mucosal erosions

    • VL – multisystem involvement, potentially fatal

Exam: Ocular

Anterior Segment

  • CL – lid or periocular ulcers, madarosis, ectropion, trichiasis, exposure keratopathy

  • ML – lid lesions similar to chalazia, basal cell carcinoma; dacryocystitis; interstitial keratitis

  • VL – same as in CL, ML; also bilateral anterior uveitis, cataract, phthisis

Posterior Segment

  • CL, ML – none

  • VL – panuveitis, RD (retinal detachment) with PVR (proliferative vitreoretinopathy), retinal hemorrhage, glaucoma

Exam: Systemic

  • CL – single/multiple papules, nodules, ulcers with raised margins and depressed center, may scab; regional lymphangitis

  • ML – mucosal erosions, perforated nasal septum, oral/pharyngeal ulcers

  • VL – hepatosplenomegaly, fever, anemia, pancytopenia, lymphadenopathy, hypergammaglobulinemia, hypoalbuminemia; post-VL dermal leishmaniasis with skin lesions involving face, trunk and genitals

Imaging

  • Fundus photograph, fundus fluorescein angiography, and optical coherence tomography: may be needed to evaluate the involvement of the posterior segment

Laboratory and Radiographic Testing

  • Biopsy of involved tissue demonstrating protozoa (amastigotes) with light microscopy (gold standard)
    • CL, ML – skin and mucosal lesions

    • VL – bone marrow, splenic aspirate with high yield (latter may have potentially lethal complications and is thus avoided)

  • VL – culture (Novy-MacNeal-Nicolle medium), deoxyribonucleic acid (DNA) PCR, and serologies (rk39 rapid test, Leishmania IgM/IgG)

Differential Diagnosis

  • CL – infections including bacterial, fungal, viral; sporotrichosis; dermatologic malignancies including basal cell carcinoma; inflammatory disease including plaque psoriasis

  • ML – granulomatosis with polyangiitis (previously Wegener’s), lethal midline granuloma, polymorphic reticulosis, lymphoma, nasopharyngeal carcinoma

  • VL – systemic infections including military tuberculosis (TB), syphilis, brucellosis, endocarditis; malignancy including lymphoma, leukemia

Treatment

  • Preventative
    • Clothing, insect repellent, avoidance of nocturnal activities

    • Early diagnosis and treatment of infected individuals who may act as reservoir

    • Control insect breeding

    • Improvement of living conditions, nutrition, immunocompromised conditions (HIV)

  • Therapy
    • Pentavalent ammonium compounds
      • Sodium stibogluconate 20 mg/kg intravenous (IV) for 20 days (CL) or 28 days (VL)

    • Liposomal amphotericin B (VL)
      • Immunocompetent – 3 mg/kg/day IV on days 1–5, 14, and 21

      • Immunocompromised – 4 mg/kg/day IV on days 1–5, 10, 17, 24, 31, and 38

    • Conventional amphotericin B deoxycholate 0.5–1 mg/kg/day, total dose 15–20 mg/kg or more

    • Miltefosine (>12 yo) 50 mg BID (30–44 kg) or TID (>45 kg) for 28 days
      • Contraindicated in pregnancy, defer conception by 5 months

    • CL – ketoconazole 600 mg/day for 28 days; itraconazole 200 mg BID for 28 days; fluconazole 200–400 mg/day for 6 weeks

    • Pentamidine, allopurinol, topical paromomycin

    • Topical steroid and cycloplegia for anterior uveitis

    • Systemic corticosteroid may help posterior ocular findings

Referral/Co-management

  • Dermatology

  • Primary Care

  • Infectious Disease

Malaria

Overview

  • Definition
    • Caused by protozoa Plasmodium and transmitted by mosquito

    • Intraerythrocytic parasite

    • Ocular involvement in 20–30%, typically severe malaria

    • Plasmodium vivax (Pv) causes milder disease than P. falciparum (Pf) which can be lethal (cerebral malaria)

  • Symptoms
    • Blurring

    • Conjunctival discoloration
      • Hemorrhage

      • Yellowing

    • Pain

    • Light sensitivity

    • Floaters

  • Laterality
    • Unilateral or bilateral

  • Course
    • Typically present only in severe malaria

  • Age of onset
    • All age groups affected

  • Gender/race
    • Any/any person in endemic area

  • Systemic association
    • Systemic malarial infection
      • Severe flu-like symptoms

      • Encephalopathy

      • May lead to convulsion, coma, or death

Exam: Ocular

Anterior Segment

  • Subconjunctival hemorrhage

  • Conjunctival yellowing

  • Anterior uveitis

Posterior Segment

  • Retinal hemorrhage, preretinal or intraretinal

  • Roth spots

  • Vitreous hemorrhage

  • Retinal edema

  • Retinal ischemia

  • Retinal vasculitis, periphlebitis (poor prognosis)

  • Papilledema and optic neuritis (poor prognosis)

Exam: Systemic

  • Fever with chills and rigor at definite intervals

  • Headache

  • Fatigue

  • Muscle ache

  • Nausea and vomiting

  • Orthostatic hypotension

  • Hepatosplenomegaly, anemia, thrombocytopenia

  • Severe cases (Pf)
    • High fever (>40 °C)

    • Tachycardia

    • Delirium

    • Cerebral malaria with diffuse symmetrical encephalopathy
      • Potentially fatal

Imaging

  • OCT: may help to locate the location of hemorrhage

  • FA: retinal vascular or optic disc leakage, occlusion; blocking defects from hemorrhage

  • VF: scotomas

Laboratory and Radiographic Testing

  • Peripheral blood smear showing protozoa (schizonts) within red blood cells (RBCs)

  • Rapid diagnostic stick tests
    • P. falciparum histidine–rich protein 2 (PfHRP2)

    • Plasmodium lactate dehydrogenase (PLDH) for pan-malaria group

Differential Diagnosis

  • Infectious – bacterial, fungal, viral, or protozoal

  • Collagen vascular disease

Malignancy

  • Heat stroke

Treatment

  • Pf
    • Quinine-based therapy
      • Quinine 1 g (600 mg base) PO, then 500 mg (300 mg base) PO 6–8 hours later, then 500 mg (300 mg base) PO at 24 hours and 48 hours after the initial dosewith

      • Pyrimethamine-sulfadoxine, or doxycycline or clindamycin

    • Artemether-lumefantrine

    • Atovaquone-proguanil

    • Mefloquine

  • Pv
    • Chloroquine with primaquine

    • Dihydroartemisinin-piperaquine with primaquine

  • P. malariae
    • Chloroquine

Referral/Co-management

  • Infectious Disease

  • Primary Care/ICU

SHAPU (Seasonal Hyperacute Panuveitis)

Overview

  • Definition
    • Seasonal severe unilateral inflammation occurring every 2 years in autumn

    • Suspected relation to tussock moth; exact cause is unknown

    • Previously called “seasonal endophthalmitis”

  • Symptoms
    • Blurring

    • White or red eye

    • Floaters

    • Lack of pain

  • Laterality
    • Unilateral

  • Course
    • Hyperacute, rapid progression to blindness/phthisis

  • Age of onset
    • Typically pediatric

  • Gender/race
    • No gender predilection

    • Only reported in Nepal

  • Systemic association
    • None

Exam: Ocular

Anterior Segment

  • Severe anterior uveitis
    • May have hypopyon/fibrinoid reaction

    • Non-granulomatous or granulomatous

  • White pupillary reflex

  • Shallow anterior chamber

  • Non-dilating pupil

  • Hypotony (malignant hypotension) very typical

  • Rapid progression to phthisis in weeks

Posterior Segment

  • Severe vitritis

Exam: Systemic

  • No findings

Imaging

  • Ultrasonography: used to evaluate the vitreous, retina and choroid

Laboratory and Radiographic Testing

  • Vitreous or aqueous tap showing Streptococcus pneumonia, Acinetobacter spp., varicella-zoster virus, and anellovirus (torque teno virus, torque teno midi, or torque teno mini virus) in some cases

  • A yet-undiagnosed infectious organism or severe allergic response to moth antigen may be involved in the etiopathogenesis

Differential Diagnosis

  • Endophthalmitis – endogenous, exogenous, traumatic

  • Acute retinal necrosis

  • Intraocular TB

  • Syphilis

  • Toxoplasmosis

  • Sarcoidosis

  • Behcet’s disease

Treatment

  • Early PPV (pars plana vitrectomy) only successful method of salvaging vision reported
    • Intraocular antibiotic, antiviral can be given after surgery

  • Supportive care with topical antibiotic, steroid, cycloplegia

Referral/Co-management

  • Microbiology

  • Infectious Disease