6. No morir de diabetes

 

[1]. Matthews, D. R., Matthews, P. C., «Banting Memorial Lecture 2010. Type 2 diabetes as an 'infectious' disease: is this the Black Death of the 21st century?», en Diabet Med., 28(1), 2011, pp. 2–9.

[2]. Centers for Disease Control and Prevention, «Number (in millions) of civilian, noninstitutionalized persons with diagnosed diabetes, United States, 1980–2011», en <http://www.cdc.gov/diabetes/statistics/prev/national/figpersons.htm>, 28 de marzo de 2013. Último acceso 3 de mayo de 2015.

[3]. Boyle, J. P., Thompson, T. J., Gregg, E. W., Barker, L. E., Williamson, D. F., «Projection of the year 2050 burden of diabetes in the US adult population: dynamic modeling of incidence, mortality, and prediabetes prevalence», en Popul Health Metr., 8, 2010, p. 29.

[4]. Centers for Disease Control and Prevention, «National Diabetes Statistics Report: Estimates of Diabetes and Its Burden in the United States, 2014», U.S. Department of Health and Human Services, Atlanta, Georgia, 2014.

[5]. Centers for Disease Control and Prevention, «Deaths: final data for 2013 table 10. Number of deaths from 113 selected causes», en National Vital Statistics Report, 64(2), 2016.

[6]. «2014 Statistics Report», Centers for Disease Control and Prevention, en <http://www.cdc.gov/diabetes/data/statistics/2014StatisticsReport.htm>, actualizado el 24 de octubre de 2014. Último acceso 3 de marzo de 2015.

[7]. Lempainen, J., Tauriainen, S., Vaarala, O., et al., «Interaction of enterovirus infection and cow's milkbased formula nutrition in type 1 diabetes-associated autoimmunity», en Diabetes Metab Res Rev., 28(2), 2012, pp. 177–185.

[8]. «2014 Statistics Report», Centers for Disease Control and Prevention, en <http://www.cdc.gov/diabetes/data/statistics/2014StatisticsReport.htm>, actualizado el 24 de octubre de 2014. Último acceso 3 de marzo de 2015.

[9]. Rachek, L. I., «Free fatty acids and skeletal muscle insulin resistance», en Prog Mol Biol Transl Sci., 121, 2014, pp. 267–292.

[10]. «2014 Statistics Report», Centers for Disease Control and Prevention, en <http://www.cdc.gov/diabetes/data/statistics/2014StatisticsReport.html>, actualizado el 24 de octubre de 2014. Último acceso 3 de marzo de 2015.

[11]. Sweeney, J. S., «Dietary factors that influence the dextrose tolerance test», en Arch Intern Med., 40(6), 1927, pp. 818–830.

[12]. Roden, M., Price, T. B., Perseghin, G., et al., «Mechanism of free fatty acid-induced insulin resistance in humans», en J Clin Invest., 97(12), 1996, pp. 2859–2865.

[13]. Roden, M., Krssak, M., Stingl, H., et al., «Rapid impairment of skeletal muscle glucose transport/phosphorylation by free fatty acids in humans», en Diabetes, 48(2), 1999, pp. 358–364.

[14]. Santomauro, A. T., Boden, G., Silva, M. E., et al., «Overnight lowering of free fatty acids with Acipimox improves insulin resistance and glucose tolerance in obese diabetic and nondiabetic subjects», en Diabetes, 48(9), 1999, pp. 1836–1841.

[15]. Krssak, M., Falk Petersen, K., Dresner, A., et al., «Intramyocellular lipid concentrations are correlated with insulin sensitivity in humans: a 1H NMR spectroscopy study», en Diabetologia, 42(1), 1999, pp. 113–116.

[16]. Lee, S., Boesch, C., Kuk, J. L., Arslanian, S., «Effects of an overnight intravenous lipid infusion on intramyocellular lipid content and insulin sensitivity in African-American versus Caucasian adolescents», en Metab Clin Exp., 62(3), 2013, pp. 417–423.

[17]. Roden, M., Krssak, M., Stingl, H., et al., «Rapid impairment of skeletal muscle glucose transport/phosphorylation by free fatty acids in humans», en Diabetes, 48(2), 1999, pp. 358–364.

[18]. Himsworth, H. P., «Dietetic factors influencing the glucose tolerance and the activity of insulin», en J Physiol (Londres), 81(1), 1934, pp. 29–48.

[19]. Tabák, A. G. 1, Herder, C., Rathmann, W., Brunner, E. J., Kivimäki, M., «Prediabetes: a high-risk state for diabetes», en Lancet, 379(9833), 2012, pp. 2279–2290.

[20]. Pratley, R. E., «The early treatment of type 2 diabetes», en Am J Med., 26(9 Suppl 1), 2013, pp. S2–S9.

[21]. Reinehr, T., «Type 2 diabetes mellitus in children and adolescents», en World J Diabetes, 4(6), 2013, pp. 270–281.

[22]. Pihoker, C., Scott, C. R., Lensing, S. Y., Cradock, M. M., Smith, J., «Non-insulin dependent diabetes mellitus in African-American youths of Arkansas», en Clin Pediatr. (Filadelfia), 37(2), 1998, pp. 97–102.

[23]. Dean, H., Flett, B., «Natural history of type 2 diabetes diagnosed in childhood: long term follow-up in young adult years», en Diabetes, 51(s1), 2002, p. A24.

[24]. Hannon, T. S., Rao, G., Arslanian, S. A., «Childhood obesity and type 2 diabetes mellitus», en Pediatrics, 116(2), 2005, pp. 473–480.

[25]. Rocchini, A. P., «Childhood obesity and a diabetes epidemic», en N Engl J Med., 346(11), 2002, pp. 854–855.

[26]. Lifshitz, F., «Obesity in children», en J Clin Res Pediatr Endocrinol., 1(2), 2008, pp. 53–60.

[27]. Must, A., Jacques, P. F., Dallal, G. E., Bajema, C. J., Dietz, W. H., «Long-term morbidity and mortality of overweight adolescents. A follow-up of the Harvard Growth Study of 1922 to 1935», en N Engl J Med., 327(19), 1992, pp. 1350–1355.

[28]. Sabaté, J., Wien, M., «Vegetarian diets and childhood obesity prevention», en Am J Clin Nutr., 91(5), 2010, pp. 1525S–1529S.

[29]. Tonstad, S., Butler, T., Yan, R., Fraser, G. E., «Type of vegetarian diet, body weight, and prevalence of type 2 diabetes», en Diabetes Care, 32(5), 2009, 791–796.

[30]. Sabaté, J., Lindsted, K. D., Harris, R. D., Sanchez, A., «Attained height of lacto-ovo vegetarian children and adolescents», en Eur J Clin Nutr., 45(1), 1991, pp. 51–58.

[31]. Sabaté, J., Wien, M., «Vegetarian diets and childhood obesity prevention», en Am J Clin Nutr., 91(5), 2010, pp. 1525S–1529S.

[32]. Cali, A. M., Caprio, S., «Prediabetes and type 2 diabetes in youth: an emerging epidemic disease?», en Curr Opin Endocrinol Diabetes Obes., 15(2), 2008, pp. 123–127.

[33]. Ginter, E., Simko, V., «Type 2 diabetes mellitus, pandemic in 21st century», en Adv Exp Med Biol., 771, 2012, pp. 42–50.

[34]. Spalding, K. L., Arner, E., Westermark, P. O., et al., «Dynamics of fat cell turnover in humans», en Nature, 453(7196), 2008, pp. 783–787.

[35]. Roden, M., «How free fatty acids inhibit glucose utilization in human skeletal muscle», en News Physiol Sci., 19, 2004, pp. 92–96.

[36]. Fraser, G. E., «Vegetarian diets: what do we know of their effects on common chronic diseases?», en Am J Clin Nutr., 89(5), 2009, pp. 1607S–1612S.

[37]. Tonstad, S., Stewart, K., Oda, K., Batech, M., Herring, R. P., Fraser, G. E., «Vegetarian diets and incidence of diabetes in the Adventist Health Study-2», en Nutr Metab Cardiovasc Dis., 23(4), 2013, pp. 292–299.

[38]. Nolan, C. J., Larter, C. Z., «Lipotoxicity: why do saturated fatty acids cause and monounsaturates protect against it?», en J Gastroenterol Hepatol., 24(5), 2009, pp. 703–706.

[39]. Evans, W. J., «Oxygen-carrying proteins in meat and risk of diabetes mellitus», en JAMA Intern Med., 173(14), 2013, pp. 1335–1336.

[40]. Egnatchik, R. A., Leamy, A. K., Jacobson, D. A., Shiota, M., Young, J. D., «ER calcium release promotes mitochondrial dysfunction and hepatic cell lipotoxicity in response to palmitate overload», en Mol Metab., 3(5), 2014, pp. 544–553.

[41]. Estadella, D., Da Penha Oller do Nascimento, C. M., Oyama, L. M., Ribeiro, E. B., Dâmaso, A. R., De Piano, A., «Lipotoxicity: effects of dietary saturated and transfatty acids», en Mediators Inflamm., 2013, 2013, p. 137579.

[42]. Perseghin, G., Scifo, P., De Cobelli, F., et al., «Intramyocellular triglyceride content is a determinant of in vivo insulin resistance in humans: a 1H-13C nuclear magnetic resonance spectroscopy assessment in off spring of type 2 diabetic parents», en Diabetes, 48(8), 1999, pp. 1600–1606.

[43]. Nolan, C. J., Larter, C. Z., «Lipotoxicity: why do saturated fatty acids cause and monounsaturates protect against it?», en J Gastroenterol Hepatol., 24(5), 2009, pp. 703–706.

[44]. Goff, L. M., Bell, J. D., So, P.W., Dornhorst, A., Frost, G. S., «Veganism and its relationship with insulin resistance and intramyocellular lipid», en Eur J Clin Nutr., 59(2), 2005, pp. 291–298.

[45]. Gojda, J., Patková, J., Jaček, M., et al., «Higher insulin sensitivity in vegans is not associated with higher mitochondrial density», en Eur J Clin Nutr., 67(12), 2013, pp. 1310–1315.

[46]. Goff, L. M., Bell, J. D., So, P. W., Dornhorst, A., Frost, G. S., «Veganism and its relationship with insulin resistance and intramyocellular lipid», en Eur J Clin Nutr., 59(2), 2005, pp. 291–298.

[47]. Papanikolaou, Y., Fulgoni, V. L., «Bean consumption is associated with greater nutrient intake, reduced systolic blood pressure, lower body weight, and a smaller waist circumference in adults: results from the National Health and Nutrition Examination Survey 1999–2002», en J Am Coll Nutr., 27(5)2008, pp. 569–576.

[48]. Mollard, R. C., Luhovyy, B. L., Panahi, S., Nunez, M., Hanley, A., Anderson, G. H., «Regular consumption of pulses for 8 weeks reduces metabolic syndrome risk factors in overweight and obese adults», en Br J Nutr., 108 Suppl 1, 2012, pp. S111–S122.

[49]. Cnop, M., Hughes, S. J., Igoillo Esteve, M., et al., «The long lifespan and low turnover of human islet beta cells estimated by mathematical modelling of lipofuscin accumulation», en Diabetologia, 53(2), 2010, pp. 321–330.

[50]. Taylor, R., «Banting Memorial lecture 2012: reversing the twin cycles of type 2 diabetes», en Diabet Med., 30(3), 2013, pp. 267–275.

[51]. Cunha, D. A., Igoillo-Esteve, M., Gurzov, E. N., et al., «Death protein 5 and p53-upregulated modulator of apoptosis mediate the endoplasmic reticulum stress-mitochondrial dialog triggering lipotoxic rodent and human β-cell apoptosis», en Diabetes, 61(11), 2012, pp. 2763–2775.

[52]. Cnop, M., Hannaert, J. C., Grupping, A. Y., Pipeleers, D. G., «Low density lipoprotein can cause death of islet beta-cells by its cellular uptake and oxidative modification», en Endocrinology, 143(9), 2002, pp. 3449–3453.

[53]. Maedler, K., Oberholzer, J., Bucher, P., Spinas, G. A., Donath, M. Y., «Monounsaturated fatty acids prevent the deleterious effects of palmitate and high glucose on human pancreatic beta-cell turnover and function», en Diabetes, 52(3), 2003, pp. 726–733.

[54]. Xiao, C., Giacca, A., Carpentier, A., Lewis, G. F., «Differential effects of monounsaturated, polyunsaturated and saturated fat ingestion on glucose-stimulated insulin secretion, sensitivity and clearance in overweight and obese, non-diabetic humans», en Diabetologia, 49(6), 2006, pp. 1371–1379.

[55]. Wang, L., Folsom, A. R., Zheng, Z. J., Pankow, J. S., Eckfeldt, J. H., «Plasma fatty acid composition and incidence of diabetes in middle-aged adults: the Atherosclerosis Risk in Communities (ARIC) Study», en Am J Clin Nutr., 78(1), 2003, pp. 91–98.

[56]. Cunha, D. A., Igoillo-Esteve, M., Gurzov, E. N., et al., «Death protein 5 and p53-upregulated modulator of apoptosis mediate the endoplasmic reticulum stress-mitochondrial dialog triggering lipotoxic rodent and human β-cell apoptosis», en Diabetes, 61(11), 2012, pp. 2763–2775.

[57]. Welch, R. W., «Satiety: have we neglected dietary non-nutrients?», en Proc Nutr Soc., 70(2), 2011, pp. 145–154.

[58]. Barnard, N. D., Cohen, J., Jenkins, D. J., et al., «A low-fat vegan diet improves glycemic control and cardiovascular risk factors in a randomized clinical trial in individuals with type 2 diabetes», en Diabetes Care, 29(8), 2006, pp. 1777–1783.

[59]. Trapp, C. B., Barnard, N. D., «Usefulness of vegetarian and vegan diets for treating type 2 diabetes», en Curr Diab Rep., 10(2), 2010, pp. 152–158.

[60]. Pratley, R. E., «The early treatment of type 2 diabetes», en Am J Med., 126(9 Suppl 1), 2013, pp. S2–S9.

[61]. Juutilainen, A., Lehto, S., Rönnemaa, T., Pyörälä, K., Laakso, M., «Type 2 diabetes as a "coronary heart disease equivalent": an 18-year prospective population-based study in Finnish subjects», en Diabetes Care, 28(12), 2005, pp. 2901–2907.

[62]. Kahleova, H., Matoulek, M., Malinska, H., et al., «Vegetarian diet improves insulin resistance and oxidative stress markers more than conventional diet in subjects with type 2 diabetes», en Diabet Med., 28(5), 2011, pp. 549–559.

[63]. Ornish, D., «Statins and the soul of medicine», en Am J Cardiol., 89(11), 2002, pp. 1286–1290.

[64]. Kahleova, H., Hrachovinova, T., Hill, M., et al., «Vegetarian diet in type 2 diabetes-improvement in quality of life, mood and eating behaviour», en Diabet Med., 30(1), 2013, pp. 127–129.

[65]. Chiu, T. H. T., Huang, H. Y., Chiu, Y. F., et al., «Taiwanese vegetarians and omnivores: dietary composition, prevalence of diabetes and IFG», en PLoS One, 9(2), 2014, p. e88547.

[66]. Chiu, T. H. T., Huang, H. Y., Chiu, Y. F., et al., «Taiwanese vegetarians and omnivores: dietary composition, prevalence of diabetes and IFG», en PLoS One, 9(2), 2014, p. e88547.

[67]. Magliano, D. J., Loh, V. H. Y., Harding, J. L., et al., «Persistent organic pollutants and diabetes: a review of the epidemiological evidence», en Diabetes Metab., 40(1), 2014, pp. 1–14.

[68]. Lee, D. H., Lee, I. K., Song, K., et al., «A strong dose-response relation between serum concentrations of per sis tent organic pollutants and diabetes: results from the National Health and Examination Survey 1999–2002», en Diabetes Care, 29(7), 2006, pp. 1638–1644.

[69]. Wu, H., Bertrand, K. A., Choi, A. L., et al., «Persistent organic pollutants and type 2 diabetes: a prospective analysis in the Nurses' Health Study and meta-analysis», en Environ Health Perspect., 121(2), 2013, pp. 153–161.

[70]. Schecter, A., Colacino, J., Haffner, D., et al., «Perfluorinated compounds, polychlorinated biphenyls, and organochlorine pesticide contamination in composite food samples from Dallas, Texas, USA», en Environ Health Perspect., 118(6), 2010, pp. 796–802.

[71]. Crinnion, W. J., «The role of persistent organic pollutants in the worldwide epidemic of type 2 diabetes mellitus and the possible connection to farmed Atlantic salmon (Salmo salar)», en Altern Med Rev., 16(4), 2011, pp. 301–313.

[72]. Lee, D. H., Lee, I. K., Song, K., et al., «A strong dose-response relation between serum concentrations of per sis tent organic pollutants and diabetes: results from the National Health and Examination Survey 1999–2002», en Diabetes Care, 29(7), 2006, pp. 1638–1644.

[73]. Crinnion, W. J., «The role of persistent organic pollutants in the worldwide epidemic of type 2 diabetes mellitus and the possible connection to farmed Atlantic salmon (Salmo salar)», en Altern Med Rev., 16(4), 2011, pp. 301–313.

[74]. Farmer, B., Larson, B. T., Fulgoni, V. L. III, et al., «A vegetarian dietary pattern as a nutrient-dense approach to weight management: an analysis of the National Health and Nutrition Examination Survey 1999–2004», en J Am Diet Assoc., 111(6), 2011, pp. 819–827.

[75]. Farmer, B., Larson, B. T., Fulgoni, V. L. III, et al., «A vegetarian dietary pattern as a nutrient-dense approach to weight management: an analysis of the National Health and Nutrition Examination Survey 1999–2004», en J Am Diet Assoc., 111(6), 2011, pp. 819–827.

[76]. Toth, M. J., Poehlman, E. T., «Sympathetic nervous system activity and resting metabolic rate in vegetarians», en Metabolism, 43(5), 1994, pp. 621–625.

[77]. Karlic, H., Schuster, D., Varga, F., et al., «Vegetarian diet affects genes of oxidative metabolism and collagen synthesis», en Ann Nutr Metab., 53(1), 2008, pp. 29–32.

[78]. Vergnaud, A. C., Norat, T., Romaguera, D., et al., «Meat consumption and prospective weight change in participants of the EPIC-PANACEA study», en Am J Clin Nutr., 92(2), 2010, pp. 398–407.

[79]. «The Action to Control Cardiovascular Risk in Diabetes Study Group», Gerstein, H. C., Miller, M. E., et al., «Effects of intensive glucose lowering in type 2 diabetes», en N Engl J Med., 358(24), 2008, pp. 2545–2559.

[80]. «The Action to Control Cardiovascular Risk in Diabetes Study Group», Gerstein, H. C., Miller, M. E., et al., «Effects of intensive glucose lowering in type 2 diabetes», en N Engl J Med., 358(24), 2008, pp. 2545–2559.

[81]. Luan, F. L., Nguyen, K., «Intensive glucose control in type 2 diabetes», en N Engl J Med., 359(14), 2008, pp. 1519–1520.

[82]. Blagosklonny, M. V., «Prospective treatment of age-related diseases by slowing down aging», en Am J Pathol., 181(4), 2012, pp. 1142–1146.

[83]. Madonna, R., Pandolfi, A., Massaro, M., et al., «Insulin enhances vascular cell adhesion molecule-1 expression in human cultured endothelial cells through a pro-atherogenic pathway mediated by p38 mitogen-activated protein-kinase», en Diabetologia, 47(3), 2004, pp. 532–536.

[84]. Lingvay, I., Guth, E., Islam, A., et al., «Rapid improvement in diabetes after gastric bypass surgery: is it the diet or surgery?», en Diabetes Care, 36(9), 2013, pp. 2741–2747.

[85]. Lingvay, I., Guth, E., Islam, A., et al., «Rapid improvement in diabetes after gastric bypass surgery: is it the diet or surgery?», en Diabetes Care, 36(9), 2013, pp. 2741–2747.

[86]. Taylor, R., «Type 2 diabetes: etiology and reversibility», en Diabetes Care, 36(4), 2013, pp. 1047–1055.

[87]. Lim, E. L., Hollingsworth, K. G., Aribisala, B. S., Chen, M. J., Mathers, J. C., Taylor, R., «Reversal of type 2 diabetes: normalisation of beta cell function in association with decreased pancreas and liver triacylglycerol», en Diabetologia, 54(10), 2011, pp. 2506–2514.

[88]. Taheri, S., Tahrani, A., Barnett, A., «Bariatric surgery: a cure for diabetes?», en Pract Diabetes Int., 26, 2009, pp. 356–358.

[89]. Vergnaud, A. C., Norat, T., Romaguera, D., et al., «Meat consumption and prospective weight change in participants of the EPIC-PANACEA study», en Am J Clin Nutr., 92(2), 2010, pp. 398–407.

[90]. Gilsing, A. M., Weijenberg, M. P., Hughes, L. A., et al., «Longitudinal changes in BMI in older adults are associated with meat consumption differentially, by type of meat consumed», en J Nutr., 142(2), 2012, pp. 340–349.

[91]. Wang, Y., Lehane, C., Ghebremeskel, K., et al., «Modern organic and broiler chickens sold for human consumption provide more energy from fat than protein», en Public Health Nutr., 13(3), 2010, pp. 400–408.

[92]. National Cattlemen’s Beef Association, Young, M. K., Redson, B. A., «New USDA data show 29 beef cuts now meet government guidelines for lean», en <http://www.beef.org/udocs/29leancuts.pdf>, 2005. Último acceso 6 de marzo de 2015.

[93]. Steven, S., Lim, E. L., Taylor, R., «Dietary reversal of type 2 diabetes motivated by research knowledge», en Diabet Med., 27(6), 2010, pp. 724–725.

[94]. Taylor, R., «Pathogenesis of type 2 diabetes: tracing the reverse route from cure to cause», en Diabetologia, 51(10), 2008, pp. 1781–1789.

[95]. American Diabetes Association, «Standards of medical care in diabetes—2015», en Diabetes Care, 38(suppl 1), 2015, pp. S1– S93.

[96]. Dunaief, D. M., Fuhrman, J., Dunaief, J. L., et al., «Glycemic and cardiovascular parameters improved in type 2 diabetes with the high nutrient density (HND) diet», en Open Journal of Preventive Medicine, 2(3), 2012, pp. 364–371.

[97]. Lim, E. L., Hollingsworth, K. G., Aribisala, B. S., Chen, M. J., Mathers, J. C., Taylor, R., «Reversal of type 2 diabetes: normalisation of beta cell function in association with decreased pancreas and liver triacylglycerol», en Diabetologia, 54(10), 2011, pp. 2506–2514.

[98]. Steven, S., Lim, E. L., Taylor, R., «Population response to information on reversibility of Type 2 diabetes», en Diabet Med., 30(4), 2013, pp. e135–e138.

[99]. Dunaief, D. M., Fuhrman, J., Dunaief, J. L., et al., «Glycemic and cardiovascular parameters improved in type 2 diabetes with the high nutrient density (HND) diet», en Open J Prev Med., 2(3), 2012, pp. 364–371.

[100]. Anderson, J. W., Ward, K., «High-carbohydrate, high-fiber diets for insulin-treated men with diabetes mellitus», en Am J Clin Nutr., 32(11), 1979, pp. 2312–2321.

[101]. Anderson, J. W., Ward, K., «High-carbohydrate, high-fiber diets for insulin-treated men with diabetes mellitus», en Am J Clin Nutr., 32(11), 1979, pp. 2312–2321.

[102]. Callaghan, B. C., Cheng, H., Stables, C. L., et al., «Diabetic neuropathy: clinical manifestations and current treatments», en Lancet Neurol., 11(6), 2012, pp. 521–534.

[103]. Said, G., «Diabetic neuropathy—a review», en Nat Clin Pract Neurol., 3(6), 2007, pp. 331–340.

[104]. Crane, M. G., Sample, C., «Regression of diabetic neuropathy with total vegetarian (vegan) diet», en J Nutr Med., 4(4), 1994, pp. 431–439.

[105]. Crane, M. G., Sample, C., «Regression of diabetic neuropathy with total vegetarian (vegan) diet», en J Nutr Med., 4(4), 1994, pp. 431–439.

[106]. Rabinowitch, I. M., «Effects of the high carbohydrate-low calorie diet upon carbohydrate tolerance in diabetes mellitus», en Can Med Assoc J., 33(2), 1935, pp. 136–144.

[107]. Newborg, B., Kempner, W., «Analysis of 177 cases of hypertensive vascular disease with papilledema; one hundred twenty-six patients treated with rice diet», en Am J Med., 19(1), 1955, pp. 33–47.

[108]. Crane, M. G., Sample, C., «Regression of diabetic neuropathy with total vegetarian (vegan) diet», en J Nutr Med., 4(4), 1994, pp. 431–439.

[109]. Crane, M. G., Sample, C., «Regression of diabetic neuropathy with total vegetarian (vegan) diet», en J Nutr Med., 4(4), 1994, pp. 431–439.

[110]. Crane, M. G., Sample, C., «Regression of diabetic neuropathy with total vegetarian (vegan) diet», en J Nutr Med., 4(4), 1994, pp. 431–439.

[111]. Crane, M. G., Zielinski, R., Aloia, R., «Cis and trans fats in omnivores, lacto-ovo vegetarians and vegans», en Am J Clin Nutr., 48, 1988, p. 920.

[112]. Tesfaye, S., Chaturvedi, N., Eaton, S. E. M., et al., «Vascular risk factors and diabetic neuropathy», en N Engl J Med., 352(4), 2005, pp. 341–350.

[113]. Newrick, P. G., Wilson, A. J., Jakubowski, J., et al., «Sural nerve oxygen tension in diabetes», en Br Med J (Clin Res Ed)., 293(6554), 1986, pp. 1053–1054.

[114]. McCarty, M. F., «Favorable impact of a vegan diet with exercise on hemorheology: implications for control of diabetic neuropathy», en Med Hypotheses, 58(6), 2002, pp. 476–486.

[115]. Kempner, W., Peschel, R. L., Schlayer, C., «Effect of rice diet on diabetes mellitus associated with vascular disease», en Postgrad Med., 24(4), 1958, pp. 359–371.

[116]. McCarty, M. F., «Favorable impact of a vegan diet with exercise on hemorheology: implications for control of diabetic neuropathy», en Med Hypotheses, 58(6), 2002, pp. 476–486.

[117]. Browning, L. M., Hsieh, S. D., Ashwell, M., «A systematic review of waist-to-height ratio as a screening tool for the prediction of cardiovascular disease and diabetes: 0,5 could be a suitable global boundary value», en Nutr Res Rev., 23(2), 2010, pp. 247–269.

[118]. Bigaard, J., Tjønneland, A., Thomsen, B. L., Overvad, K., Heitmann, B. L., Sørensen, T. I., «Waist circumference, BMI, smoking, and mortality in middle-aged men and women», en Obes Res., 11(7), 2003, pp. 895–903.

[119]. Bigaard, J., Tjønneland, A., Thomsen, B. L., Overvad, K., Heitmann, B. L., Sørensen, T. I., «Waist circumference, BMI, smoking, and mortality in middle-aged men and women», en Obes Res., 11(7), 2003, pp. 895–903.

[120]. Leitzmann, M. F., Moore, S. C., Koster, A., et al., «Waist circumference as compared with body-mass index in predicting mortality from specific causes», en PLoS One, 6(4), 2011, p. e18582.

[121]. Browning, L. M., Hsieh, S. D., Ashwell, M., «A systematic review of waist-to-height ratio as a screening tool for the prediction of cardiovascular disease and diabetes: 0,5 could be a suitable global boundary value», en Nutr Res Rev., 23(2), 2010, pp. 247–269.

[122]. Centers for Disease Control and Prevention, «National Diabetes Statistics Report: Estimates of Diabetes and Its Burden in the United States, 2014», U.S. Department of Health and Human Services Atlanta, Georgia, en <http://www.cdc.gov/diabetes/data/statistics/2014StatisticsReport.html>, actualizado el 24 de octubre de 2014. Último acceso 6 de marzo de 2015.

[123]. Nathan, D. M., Davidson, M. B., Defronzo, R. A., et al., «Impaired fasting glucose and impaired glucose tolerance: implications for care», en Diabetes Care, 30(3), 2007, pp. 753–759.

[124]. Karve, A., Hayward, R. A., «Prevalence, diagnosis, and treatment of impaired fasting glucose and impaired glucose tolerance in nondiabetic U.S. adults», en Diabetes Care, 33(11), 2010, pp. 2355–2359.

[125]. Cardona Morrell, M., Rychetnik, L., Morrell, S. L., Espinel, P. T., Bauman, A., «Reduction of diabetes risk in routine clinical practice: are physical activity and nutrition interventions feasible and are the outcomes from reference trials replicable? A systematic review and meta-analysis», en BMC Public Health, 10, 2010, pp. 653.

[126]. Holman, H., «Chronic disease—the need for a new clinical education», en JAMA, 292(9), 2004, pp. 1057–1059.

[127]. Institute of Medicine, «Crossing the Quality Chasm: A New Health System for the 21st Century», The National Academies Press, Washington, D. C., 213, 2001, en <http://www.iom.edu/Reports/2001/Crossing-the-Quality-Chasm-A-New-Health-System-for-the-21st-Century.aspx>.

 

 

7. No morir de hipertensión arterial

 

[1]. Lozano, R., Naghavi, M., Foreman, K., et al., «Global and regional mortality from 235 causes of death for 20 age groups in 1990 and 2010: a systematic analysis for the Global Burden of Disease Study 2010», en Lancet, 380(9859), 2012, pp. 2095–2128.

[2]. Das, P., Samarasekera, U., «The story of GBD 2010: a “super-human” effort», en Lancet, 380 (9859), 2012, pp. 2067–2070.

[3]. Lim, S. S., Vos, T., Flaxman, A. D., et al., «A comparative risk assessment of burden of disease and injury attributable to 67 risk factors and risk factor clusters in 21 regions, 1990–2010: a systematic analysis for the Global Burden of Disease Study 2010», en Lancet, 380(9859), 2012, pp. 2224–2260.

[4]. Lim, S. S., Vos, T., Flaxman, A. D., et al., «A comparative risk assessment of burden of disease and injury attributable to 67 risk factors and risk factor clusters in 21 regions, 1990–2010: a systematic analysis for the Global Burden of Disease Study 2010», en Lancet, 380(9859), 2012, pp. 2224–2260.

[5]. Lim, S. S., Vos, T., Flaxman, A. D., et al., «A comparative risk assessment of burden of disease and injury attributable to 67 risk factors and risk factor clusters in 21 regions, 1990–2010: a systematic analysis for the Global Burden of Disease Study 2010», en Lancet, 380(9859), 2012, pp. 2224–2260.

[6]. Bromfield, S., Muntner, P., «High blood pressure: the leading global burden of disease risk factor and the need for worldwide prevention programs», en Curr Hypertens Rep., 15(3), 2013, pp. 134–136.

[7]. Lim, S. S., Vos, T., Flaxman, A. D., et al., «A comparative risk assessment of burden of disease and injury attributable to 67 risk factors and risk factor clusters in 21 regions, 1990–2010: a systematic analysis for the Global Burden of Disease Study 2010», en Lancet, 380(9859), 2012, pp. 2224–2260.

[8]. American Heart Association, «Understanding Blood Pressure Readings», en<http://www.heart.org/HEARTORG/Conditions/HighBloodPressure/ AboutHighBloodPressure/Understanding-Blood-Pressure-Readings_ UCM_301764_Article.jsp>, 11 de marzo de 2015. Último acceso 11 de marzo de 2015.

[9]. Go, A. S., Bauman, M. A., Coleman King, S. M., et al., «An effective approach to high blood pressure control: a science advisory from the American Heart Association, the American College of Cardiology, and the Centers for Disease Control and Prevention», en J Am Coll Cardiol., 63(12), 2014, pp. 1230–1238.

[10]. Nwankwo, T., Yoon, S. S., Burt, V., Gu, Q., «Hypertension among adults in the United States: National Health and Nutrition Examination Survey, 2011–2012», en NCHS Data Brief., 133, 2013, pp. 1–8.

[11]. Walker, A. R., Walker, B. F., «High high-density-lipoprotein cholesterol in African children and adults in a population free of coronary heart diseae», en Br Med J., 2(6148), 1978, pp. 1336–1337.

[12]. Donnison, C. P., «Blood pressure in the African native», en Lancet, 213(5497), 1929, pp. 6–7.

[13]. Macmahon, S., Neal, B., Rodgers, A., «Hypertension—time to move on», en Lancet, 365(9464), 2005, pp. 1108–1109.

[14]. Law, M. R., Morris, J. K., Wald, N. J., «Use of blood pressure lowering drugs in the prevention of cardiovascular disease: meta-analysis of 147 randomised trials in the context of expectations from prospective epidemiological studies», en BMJ, 338, 2009, p. b1665.

[15]. Donnison, C. P., «Blood pressure in the African native», en Lancet, 213(5497), 1929, pp. 6–7.

[16]. Lim, S. S., Vos, T., Flaxman, A. D., et al., «A comparative risk assessment of burden of disease and injury attributable to 67 risk factors and risk factor clusters in 21 regions, 1990–2010: a systematic analysis for the Global Burden of Disease Study 2010», en Lancet, 380(9859), 2012, pp. 2224–2260.

[17]. Lim, S. S., Vos, T., Flaxman, A. D., et al., «A comparative risk assessment of burden of disease and injury attributable to 67 risk factors and risk factor clusters in 21 regions, 1990–2010: a systematic analysis for the Global Burden of Disease Study 2010», en Lancet, 380(9859), 2012, pp. 2224–2260.

[18]. Karppanen, H., Mervaala, E., «Sodium intake and hypertension», en Prog Cardiovasc Dis., 49(2), 2006, pp. 59–75.

[19]. Delahaye, F., «Should we eat less salt?», en Arch Cardiovasc Dis., 106(5), 2013, pp. 324–332.

[20]. Jenkins, D. J., Kendall, C. W., «The garden of Eden: plant-based diets, the genetic drive to store fat and conserve cholesterol, and implications for epidemiology in the 21st century», en Epidemiology, 17(2), 2006, pp. 128–130.

[21]. Roberts, W. C., «High salt intake, its origins, its economic impact, and its effect on blood pressure», en Am J Cardiol., 88(11), 2001, pp. 1338–1346.

[22]. Roberts, W. C., «High salt intake, its origins, its economic impact, and its effect on blood pressure», en Am J Cardiol., 88(11), 2001, pp. 1338–1346.

[23]. Celermajer, D. S., Neal, B., «Excessive sodium intake and cardiovascular disease: a-salting our vessels», en J Am Coll Cardiol., 61(3), 2013, pp. 344–345.

[24]. Whelton, P. K., Appel, L. J., Sacco, R. L., et al., «Sodium, blood pressure, and cardiovascular disease: further evidence supporting the American Heart Association sodium reduction recommendations», en Circulation, 126(24), 2012, pp. 2880–2889.

[25]. Centers for Disease Control and Prevention, «Sodium intake among adults - United States, 2005–2006», en MMWR Morb Mortal Wkly Rep., 59(24), 2010, pp. 746–749.

[26]. Beaglehole, R., Bonita, R., Horton, R., et al., «Priority actions for the non-communicable disease crisis», en Lancet, 377(9775), 2011, pp. 1438–1447.

[27]. Law, M. R., Frost, C. D., Wald, N. J., «By how much does dietary salt reduction lower blood pressure? III—Analysis of data from trials of salt reduction», en BMJ, 302(6780), 1991, pp. 819–824.

[28]. Bibbins-Domingo, K., Chertow, G. M., Coxson, P. G., et al., «Projected effect of dietary salt reductions on future cardiovascular disease», en N Engl J Med., 362(7), 18 de febrero de 2010, pp. 590–599.

[29]. MacGregor, G. A., Markandu, N. D., Best, F. E., et al., «Double-blind randomised crossover trial of moderate sodium restriction in essential hypertension», en Lancet, 1(8268), 1982, pp. 351–355.

[30]. MacGregor, G. A., Markandu, N. D., Sagnella, G. A., Singer, D. R., Cappuccio, F. P., «Double-blind study of three sodium intakes and long-term effects of sodium restriction in essential hypertension», en Lancet, 2(8674), 1989, pp. 1244–1247.

[31]. MacGregor, G. A., Markandu, N. D., Sagnella, G. A., Singer, D. R., Cappuccio, F. P., «Double-blind study of three sodium intakes and long-term effects of sodium restriction in essential hypertension», en Lancet, 2(8674), 1989, pp. 1244–1247.

[32]. Rudelt, A., French, S., Harnack, L., «Fourteen-year trends in sodium content of menu off erings at eight leading fast-food restaurants in the USA», en Public Health Nutr., 17(8), 2014, pp. 1682–1688.

[33]. Suckling, R. J., He, F. J., Markandu, N. D., MacGregor, G. A., «Dietary salt influences postprandial plasma sodium concentration and systolic blood pressure», en Kidney Int., 81(4), 2012, pp. 407–411.

[34]. He, F. J., Li, J., MacGregor, G. A., «Effect of longer term modest salt reduction on blood pressure: Cochrane systematic review and meta-analysis of randomised trials», en BMJ, 346, 2013, p. f1325.

[35]. Celermajer, D. S., Neal, B., «Excessive sodium intake and cardiovascular disease: a-salting our vessels», en J Am Coll Cardiol., 61(3), 2013, pp. 344–345.

[36]. Oliver, W. J., Cohen, E. L., Neel, J. V., «Blood pressure, sodium intake, and sodium related hormones in the Yanomamo Indians, a “no-salt” culture», en Circulation, 52(1), 1975, pp. 146–151.

[37]. Mancilha-Carvalho, J. de J., De Souza e Silva, N. A., «The Yanomami Indians in the INTERSALT Study», en Arq Bras Cardiol., 80(3), 2003, pp. 289–300.

[38]. Celermajer, D. S., Neal, B., «Excessive sodium intake and cardiovascular disease: a-salting our vessels», en J Am Coll Cardiol., 61(3), 2013, pp. 344–345.

[39]. Mancilha-Carvalho, J. de J., De Souza e Silva, N. A., «The Yanomami Indians in the INTERSALT Study», en Arq Bras Cardiol., 80(3), 2003, pp. 289–300.

[40]. Mancilha-Carvalho, J. de J., Crews, D. E., «Lipid profiles of Yanomamo Indians of Brazil», en Prev Med., 19(1), 1990, pp. 66–75.

[41]. Kempner, W., «Treatment of heart and kidney disease and of hypertensive and arteriosclerotic vascular disease with the rice diet», en Ann Intern Med., 31(5), 1949, pp. 821–856.

[42]. Klemmer, P., Grim, C. E., Luft, F. C., «Who and what drove Walter Kempner? The rice diet revisited», en Hypertension, 64(4), 2014, pp. 684–688.

[43]. Kempner, W., «Treatment of heart and kidney disease and of hypertensive and arteriosclerotic vascular disease with the rice diet», en Ann Intern Med., 31(5), 1949, pp. 821–856.

[44]. Roberts, W. C., «High salt intake, its origins, its economic impact, and its effect on blood pressure», en Am J Cardiol., 88(11), 2001, pp. 1338–1346.

[45]. Dickinson, K. M., Clifton, P. M., Keogh, J. B., «Endothelial function is impaired after a high-salt meal in healthy subjects», en Am J Clin Nutr., 93(3), 2011, pp. 500–505.

[46]. DuPont, J. J., Greaney, J. L., Wenner, M. M., et al., «High dietary sodium intake impairs endotheliumdependent dilation in healthy salt-resistant humans», en J Hypertens., 31(3), 2013, pp. 530–536.

[47]. Dickinson, K. M., Clifton, P. M., Keogh, J. B., «Endothelial function is impaired after a high-salt meal in healthy subjects», en Am J Clin Nutr., 93(3), 2011, pp. 500–505.

[48]. Greaney, J. L., DuPont, J. J., Lennon-Edwards, S. L., Sanders, P. W., Edwards, D. G., Farquhar, W. B., «Dietary sodium loading impairs microvascular function in de pen dent of blood pressure in humans: role of oxidative stress», en J Physiol (Londres), 590(Pt 21), 2012, pp. 5519–5528.

[49]. Jablonski, K. L., Racine, M. L., Geolfos, C. J., et al., «Dietary sodium restriction reverses vascular endothelial dysfunction in middle-aged/older adults with moderately elevated systolic blood pressure», en J Am Coll Cardiol., 61(3), 2013, pp. 335–343.

[50]. McCord, J. M., «Analysis of superoxide dismutase activity», en Curr Protoc Toxicol., Chapter 7, 1999, Unit 7.3.

[51]. Dickinson, K. M., Clifton, P. M., Burrell, L. M., Barrett, P. H. R., Keogh, J. B., «Postprandial effects of a high salt meal on serum sodium, arterial stiff ness, markers of nitric oxide production and markers of endothelial function», en Atherosclerosis, 232(1), 2014, pp. 211–216.

[52]. Huang Ti Nei Ching Su Wua [The Yellow Emperor’s Classic of Internal Medicine] (Trad. de Veith, I.) University of California Press, Oakland, California, 1972, p. 141.

[53]. Hanneman, R. L., Satin, M., «Comments to the Dietary Guidelines Committee on behalf of the Salt Institute. Comment ID: 000447», 23 de abril de 2009.

[54]. «Vital signs: food categories contributing the most to sodium consumption—United States, 2007–2008», en MMWR Morb Mortal Wkly Rep., 61(5), 2012, pp. 92–98.

[55]. Miller, G. D., «Comments to the Dietary Guidelines Committee on behalf of the National Dairy Council», 27 de julio de 2009.

[56]. Roberts, W. C., «High salt intake, its origins, its economic impact, and its effect on blood pressure», en Am J Cardiol., 88(11), 2001, pp. 1338–1346.

[57]. MacGregor, G., De Wardener, H. E., «Salt, blood pressure and health», en Int J Epidemiol., 31(2), 2002, pp. 320–327.

[58]. Appel, L. J., Anderson, C. A. M., «Compelling evidence for public health action to reduce salt intake», en N Engl J Med., 362(7), 2010, pp. 650–652.

[59]. Roberts, W. C., «High salt intake, its origins, its economic impact, and its effect on blood pressure», en Am J Cardiol., 88(11), 2001, pp. 1338–1346.

[60]. «Buying this chicken?», en Consum Rep., 7 de junio de 2008.

[61]. Drewnowski, A., Rehm, C. D., «Sodium intakes of US children and adults from foods and beverages by location of origin and by specific food source», en Nutrients, 5(6), 2013, pp. 1840–1855.

[62]. U.S. Department of Agriculture, Agricultural Research Service, 2014, USDA National Nutrient Database for Standard Reference, Release 27, «Pizza Hut 14” pepperoni pizza, pan crust», en <http://ndb.nal.usda.gov/ndb/foods/show/ 6800>. Último acceso 22 de marzo de 2015.

[63]. Drewnowski, A., Rehm, C. D., «Sodium intakes of US children and adults from foods and beverages by location of origin and by specific food source», en Nutrients, 5(6), 2013, pp. 1840–1855.

[64]. Blais, C. A., Pangborn, R. M., Borhani, N. O., Ferrell, M. F., Prineas, R. J., Laing, B., «Effect of dietary sodium restriction on taste responses to sodium chloride: a longitudinal study», en Am J Clin Nutr., 44(2), 1986, pp. 232–243.

[65]. Tucker, R. M., Mattes, R. D., «Are free fatty acids eff ective taste stimuli in humans?, presentado en el simposio “The Taste for Fat: New Discoveries on the Role of Fat in Sensory Perception, Metabolism, Sensory Plea sure and Beyond”, realizado en el Institute of Food Technologists 2011 Annual Meeting, Nueva Orleans, Luisiana, 12 de junio de 2011», en J Food Sci., 77(3), 2012, pp. S148–S151.

[66]. Grieve, F. G., Vander Weg, M. W., «Desire to eat high- and low-fat foods following a low-fat dietary intervention», en J Nutr Educ Behav., 35(2), 2003, pp. 98–102.

[67]. Stewart, J. E., Newman, L. P., Keast, R. S., «Oral sensitivity to oleic acid is associated with fat intake and body mass index», en Clin Nutr., 30(6), 2011, pp. 838–844.

[68]. Stewart, J. E., Keast, R. S., «Recent fat intake modulates fat taste sensitivity in lean and overweight subjects», en Int J Obes. (Londres), 36(6), 2012, pp. 834–842.

[69]. Roberts, W. C., «High salt intake, its origins, its economic impact, and its effect on blood pressure», en Am J Cardiol., 88(11), 2001, pp. 1338–1346.

[70]. Newson, R. S., Elmadfa, I., Biro, G., et al., «Barriers for progress in salt reduction in the general population. An international study», en Appetite, 71, 2013, pp. 22–31.

[71]. Cappuccio, F. P., Capewell, S., Lincoln, P., McPherson, K., «Policy options to reduce population salt intake», en BMJ, 343, 2011, p. d4995.

[72]. Toldrá, F., Barat, J. M., «Strategies for salt reduction in foods», en Recent Pat Food Nutr Agric., 4(1), 2012, pp. 19–25.

[73]. Lin, B.-H., Guthrie, J., «Nutritional Quality of Food Prepared at Home and Away from Home, 1977–2008», USDA, Economic Research Service, diciembre de 2012.

[74]. Newson, R. S., Elmadfa, I., Biro, G., et al., «Barriers for progress in salt reduction in the general population. An international study», en Appetite, 71, 2013, pp. 22–31.

[75]. Roberts, W. C., «High salt intake, its origins, its economic impact, and its effect on blood pressure», en Am J Cardiol., 88(11), 2001, pp. 1338–1346.

[76]. U.S. Department of Agriculture and U.S. Department of Health and Human Services, Dietary Guidelines for Americans, 2010. 7th Edition, US Government Printing Office, Washington, D. C., diciembre de 2010.

[77]. Karppanen, H., Mervaala, E., «Sodium intake and hypertension», en Prog Cardiovasc Dis., 49(2), 2006, pp. 59–75.

[78]. Law, M. R., Morris, J. K., Wald, N. J., «Use of blood pressure lowering drugs in the prevention of cardiovascular disease: meta-analysis of 147 randomised trials in the context of expectations from prospective epidemiological studies», en BMJ, 338, 2009, p. b1665.

[79]. Tighe, P., Duthie, G., Vaughan, N., et al., «Effect of increased consumption of whole-grain foods on blood pressure and other cardiovascular risk markers in healthy middle-aged persons: a randomized controlled trial», en Am J Clin Nutr., 92(4), 2010, pp. 733–740.

[80]. Diaconu, C. C., Balaceanu, A., Bartos, D., «Diuretics, first-line antihypertensive agents: are they always safe in the elderly?», en Rom J Intern Med., 52(2), 2014, pp. 87–90.

[81]. Li, C. I., Daling, J. R., Tang, M. T., Haugen, K. L., Porter, P. L., Malone, K. E., «Use of antihypertensive medications and breast cancer risk among women aged 55 to 74 years», en JAMA Intern Med., 173(17), 2013, pp. 1629–1637.

[82]. Kaiser, E. A., Lotze, U., Schiser, H. H., «Increasing complexity: which drug class to choose for treatment of hypertension in the elderly?», en Clin Interv Aging., 9, 2014, pp. 459–475.

[83]. Rasmussen, E. R., Mey, K., Bygum, A., «Angiotensin-converting enzyme inhibitor-induced angioedema—a dangerous new epidemic», en Acta Derm Venereol., 94(3), 2014, pp. 260–264.

[84]. Tinetti, M. E., Han, L., Lee, D. S., et al., «Antihypertensive medications and serious fall injuries in a nationally representative sample of older adults», en JAMA Intern Med., 174(4), 2014, pp. 588–595.

[85]. Ye, E. Q., Chacko, S. A., Chou, E. L., Kugizaki, M., Liu, S., «Greater whole-grain intake is associated with lower risk of type 2 diabetes, cardiovascular disease, and weight gain», en J Nutr., 142(7), 2012, pp. 1304–1313.

[86]. Aune, D., Chan, D. S., Lau, R., et al., «Dietary fibre, whole grains, and risk of colorectal cancer: systematic review and dose-response meta-analysis of prospective studies», en BMJ, 343, 2011, p. d6617.

[87]. Tighe, P., Duthie, G., Vaughan, N., et al., «Effect of increased consumption of whole-grain foods on blood pressure and other cardiovascular risk markers in healthy middle-aged persons: a randomized controlled trial», en Am J Clin Nutr., 92(4), 2010, pp. 733–740.

[88]. Sun, Q., Spiegelman, D., Van Dam, R. M., et al., «White rice, brown rice, and risk of type 2 diabetes in US men and women», en Arch Intern Med., 170(11), 2010, pp. 961–969.

[89]. Ye, E.Q., Chacko, S. A., Chou, E. L., Kugizaki, M., Liu, S., «Greater whole-grain intake is associated with lower risk of type 2 diabetes, cardiovascular disease, and weight gain», en J Nutr., 142(7), 2012, pp. 1304–1313.

[90]. Mellen, P. B., Liese, A. D., Tooze, J. A., Vitolins, M. Z., Wagenknecht, L. E., Herrington, D. M., «Wholegrain intake and carotid artery atherosclerosis in a multiethnic cohort: the Insulin Resistance Atherosclerosis Study», en Am J Clin Nutr., 85(6), 2007, pp. 1495–1502.

[91]. Erkkilä, A. T., Herrington, D. M., Mozaffarian, D., et al., «Cereal fiber and whole-grain intake are associated with reduced progession of coronary-artery atherosclerosis in postmenopausal women with coronary artery disease», en Am Heart J., 150(1), 2005, pp. 94–101.

[92]. Go, A. S., Bauman, M. A., Coleman King, S. M., et al., «An effective approach to high blood pressure control: a science advisory from the American Heart Association, the American College of Cardiology, and the Centers for Disease Control and Prevention», en J Am Coll Cardiol., 63(12), 2014, pp. 1230–1238.

[93]. Mahmud, A., Feely, J., «Low-dose quadruple antihypertensive combination: more efficacious than individual agents—a preliminary report», en Hypertension, 49(2), 2007, pp. 272–275.

[94]. Kronish, I. M., Woodward, M., Sergie, Z., Ogedegbe, G., Falzon, L., Mann, D. M., «Meta-analysis: impact of drug class on adherence to antihypertensives», en Circulation, 123(15), 2011, pp. 1611–1621.

[95]. Messerli, F. H., Bangalore, S., «Half a century of hydrochlorothiazide: facts, fads, fiction, and follies», en Am J Med., 124(10), 2011, pp. 896–899.

[96]. Law, M. R., Morris, J. K., Wald, N. J., «Use of blood pressure lowering drugs in the prevention of cardiovascular disease: meta-analysis of 147 randomised trials in the context of expectations from prospective epidemiological studies», en BMJ, 338, 2009, p. b1665.

[97]. Donnison, C. P., «Blood pressure in the African native», en Lancet, 213(5497), 1929, pp. 6–7.

[98]. Morse, W. R., McGill, M. D., Beh, Y. T., «Blood pressure amongst aboriginal ethnic groups of Szechwan Province, West China», en Lancet, 229(5929), 1937, 966–968.

[99]. Sacks, F. M., Kass, E. H., «Low blood pressure in vegetarians: effects of specific foods and nutrients», en Am J Clin Nutr., 48(3 Suppl), 1988, pp. 795–800.

[100]. Go, A. S., Bauman, M. A., Coleman King, S. M., et al., «An effective approach to high blood pressure control: a science advisory from the American Heart Association, the American College of Cardiology, and the Centers for Disease Control and Prevention», en J Am Coll Cardiol., 63(12), 2014, pp. 1230–1238.

[101]. Sharma, A. M., Schorr, U., «Dietary patterns and blood pressure», en N Engl J Med., 337(9), 1997, p. 637.

[102]. Chen, Q., Turban, S., Miller, E. R., Appel, L. J., «The effects of dietary patterns on plasma renin activity: results from the Dietary Approaches to Stop Hypertension trial», en J Hum Hypertens., 26(11), 2012, pp. 664–669.

[103]. Sacks, F. M., Rosner, B., Kass, E. H., «Blood pressure in vegetarians», en Am J Epidemiol., 100(5), 1974, pp. 390–398.

[104]. Donaldson, A. N., «The relation of protein foods to hypertension», en Cal West Med., 24(3), 1926, pp. 328–331.

[105]. Appel, L. J., Brands, M. W., Daniels, S. R., et al., «Dietary approaches to prevent and treat hypertension: a scientific statement from the American Heart Association», en Hypertension, 47(2), 2006, pp. 296–308.

[106]. Sacks, F. M., Obarzanek, E., Windhauser, M. M., et al., «Rationale and design of the Dietary Approaches to Stop Hypertension trial (DASH). A multicenter controlled-feeding study of dietary patterns to lower blood pressure», en Ann Epidemiol., 5(2), 1995, pp. 108–118.

[107]. Karanja, N. M., Obarzanek, E., Lin, P. H., et al., «Descriptive characteristics of the dietary patterns used in the Dietary Approaches to Stop Hypertension trial. DASH Collaborative Research Group», en J Am Diet Assoc., 99(8 Suppl), 1999, pp. S19–S27.

[108]. Sacks, F. M., Kass, E. H., «Low blood pressure in vegetarians: effects of specific foods and nutrients», en Am J Clin Nutr., 48(3 Suppl), 1988, pp. 795–800.

[109]. De Paula, T. P., Steemburgo, T., De Almeida, J. C., Dall’Alba, V., Gross, J. L., de Azevedo, M. J., «The role of Dietary Approaches to Stop Hypertension (DASH) diet food groups in blood pressure in type 2 diabetes», en Br J Nutr., 108(1), 2012, pp. 155–162.

[110]. Yokoyama, Y., Nishimura, K., Barnard, N. D., et al., «Vegetarian diets and blood pressure: a metaanalysis», en JAMA Intern Med., 174(4), 2014, pp. 577–587.

[111]. Le, L. T., Sabaté, J., «Beyond meatless, the health effects of vegan diets: findings from the Adventist cohorts», en Nutrients, 6(6), 2014, pp. 2131–2147.

[112]. Fraser, G. E., «Vegetarian diets: what do we know of their effects on common chronic diseases?», en Am J Clin Nutr., 89(5), 2009, pp. 1607S–1612S.

[113]. Tonstad, S., Stewart, K., Oda, K., Batech, M., Herring, R. P., Fraser, G. E., «Vegetarian diets and incidence of diabetes in the Adventist Health Study-2», en Nutr Metab Cardiovasc Dis., 23(4), 2013, pp. 292–299.

[114]. Fraser, G. E., «Vegetarian diets: what do we know of their effects on common chronic diseases?», en Am J Clin Nutr., 89(5), 2009, pp. 1607S–1612S.

[115]. Fontana, L., Meyer, T. E., Klein, S., Holloszy, J. O., «Long-term low-calorie low-protein vegan diet and endurance exercise are associated with low cardiometabolic risk», en Rejuvenation Res., 10(2), 2007, pp. 225–234.

[116]. Rodriguez-Leyva, D., Weighell, W., Edel, A. L., et al., «Potent antihypertensive action of dietary flaxseed in hypertensive patients», en Hypertension, 62(6), 2013, pp. 1081–1089.

[117]. Cornelissen, V. A., Buys, R., Smart, N. A., «Endurance exercise beneficially affects ambulatory blood pressure: a systematic review and meta-analysis», en J Hypertens., 31(4), 2013, pp. 639–648.

[118]. Geleijnse, J. M., «Relation of raw and cooked vegetable consumption to blood pressure: the INTERMAP study», en J Hum Hypertens., 28(6), 2014, pp. 343–344.

[119]. Jayalath, V. H., De Souza, R. J., Sievenpiper, J. L., et al., «Effect of dietary pulses on blood pressure: a systematic review and meta-analysis of controlled feeding trials», en Am J Hypertens., 27(1), 2014, pp. 56–64.

[120]. Chiva-Blanch, G., Urpi Sarda, M., Ros, E., et al., «Dealcoholized red wine decreases systolic and diastolic blood pressure and increases plasma nitric oxide: short communication», en Circ Res., 111(8), 2012, pp. 1065–1068.

[121]. Figueroa, A., Sanchez-Gonzalez, M. A., Wong, A., Arjmandi, B. H., «Watermelon extract supplementation reduces ankle blood pressure and carotid augmentation index in obese adults with prehypertension or hypertension», en Am J Hypertens., 25(6), 2012, pp. 640–643.

[122]. Gammon, C. S., Kruger, R., Brown, S. J., Conlon, C. A., von Hurst, P. R., «Stone house W. Daily kiwifruit consumption did not improve blood pressure and markers of cardiovascular function in men with hypercholesterolemia», en Nutr Res., 34(3), 2014, pp. 235–240.

[123]. Anderson, J. W., Weiter, K. M., Christian, A. L., Ritchey, M. B., Bays, H. E., «Raisins compared with other snack effects on glycemia and blood pressure: a randomized, controlled trial», en Postgrad Med., 126(1), 2014, pp. 37–43.

[124]. Akhtar, S., Ismail, T., Riaz, M., «Flaxseed—a miraculous defense against some critical maladies», en Pak J Pharm Sci., 26(1), 2013, pp. 199–208.

[125]. Rodriguez-Leyva, D., Weighell, W., Edel, A. L., et al., «Potent antihypertensive action of dietary flaxseed in hypertensive patients», en Hypertension, 62(6), 2013, pp. 1081–1089.

[126]. Ninomiya, T., Perkovic, V., Turnbull, F., et al., «Blood pressure lowering and major cardiovascular events in people with and without chronic kidney disease: meta-analysis of randomised controlled trials», en BMJ, 347, 2013, p. f5680.

[127]. Goyal, A., Sharma, V., Upadhyay, N., Gill, S., Sihag, M., «Flax and flaxseed oil: an ancient medicine & modern functional food», en J Food Sci Technol., 51(9), 2014, pp. 1633–1653.

[128]. Carlsen, M. H., Halvorsen, B. L., Holte, K., et al., «The total antioxidant content of more than 3100 foods, beverages, spices, herbs and supplements used worldwide», en Nutr J., 9, 2010, p. 3.

[129]. Frank, T., Netzel, G., Kammerer, D. R., et al., «Consumption of Hibiscus sabdariff a L. aqueous extract and its impact on systemic antioxidant potential in healthy subjects», en J Sci Food Agric., 92(10), 2012, pp. 2207–2218.

[130]. Chang, H. C., Peng, C. H., Yeh, D. M., Kao, E. S., Wang, C. J., «Hibiscus sabdariffa extract inhibits obesity and fat accumulation, and improves liver steatosis in humans», en Food Funct., 5(4), 2014, pp. 734–739.

[131]. Mozaffari-Khosravi, H., Jalali-Khanabadi, B. A., Afkhami-Ardekani, M., Fatehi, F., «Effects of sour tea (Hibiscus sabdariff a) on lipid profile and lipoproteins in patients with type II diabetes», en J Altern Complement Med., 15(8), 2009, pp. 899–903.

[132]. Aziz, Z., Wong, S. Y., Chong, N. J., «Effects of Hibiscus sabdariffa L. on serum lipids: a systematic review and meta-analysis», en J Ethnopharmacol., 150(2), 2013, pp. 442–450.

[133]. Lin, T.-L., Lin, H.-H., Chen, C.-C., et al., «Hibiscus sabdariffa extract reduces serum cholesterol in men and women», en Nutr Res., 27, 2007, pp. 140– 145.

[134]. Hopkins, A. L., Lamm, M. G., Funk, J. L., Ritenbaugh, C., «Hibiscus sabdariffa L. in the treatment of hypertension and hyperlipidemia: a comprehensive review of animal and human studies», en Fitoterapia, 85, 2013, pp. 84–94.

[135]. McKay, D. L., Chen, C. Y., Saltzman, E., Blumberg, J. B., «Hibiscus sabdariffa L. tea (tisane) lowers blood pressure in prehypertensive and mildly hypertensive adults», en J Nutr., 140(2), 2010, pp. 298–303.

[136]. Chobanian, A. V., Bakris, G. L., Black, H. R., et al., «Seventh report of the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure», en Hypertension, 42(6), 2003, pp. 1206–1252.

[137]. McKay, D. L., Chen, C. Y., Saltzman, E., Blumberg, J. B., «Hibiscus sabdariffa L. tea (tisane) lowers blood pressure in prehypertensive and mildly hypertensive adults», en J Nutr., 140(2), 2010, pp. 298–303.

[138]. Herrera-Arellano, A., Flores-Romero, S., Chávez-Soto, M. A., Tortoriello, J., «Effectiveness and tolerability of a standardized extract from Hibiscus sabdariffa in patients with mild to moderate hypertension: a controlled and randomized clinical trial», en Phytomedicine, 11(5), 2004, pp. 375–382.

[139]. US Food and Drug Administration, «CAPOTEN® (Captopril Tablets, USP)», en <http://www.accessdata.fda.gov/drugsatfda_docs/label/2012/01834 3s084lbl.pdf>. Último acceso 19 de marzo de 2015.

[140]. Hendricks, J. L., Marshall, T. A., Harless, J. D., Hogan, M. M., Qian, F., Wefel, J. S., «Erosive potentials of brewed teas», en Am J Dent., 26(5), 2013, pp. 278–282.

[141]. Malik, J., Frankova, A., Drabek, O., Szakova, J., Ash, C., Kokoska, L., «Aluminium and other elements in selected herbal tea plant species and their infusions», en Food Chem., 139(1–4), 2013, pp. 728–734.

[142]. Förstermann, U., «Janus-faced role of endothelial NO synthase in vascular disease: uncoupling of oxygen reduction from NO synthesis and its pharmacological reversal», en Biol Chem., 387(12), 2006, pp. 1521–1533.

[143]. Franzini, L., Ardigò, D., Valtueña, S., et al., «Food selection based on high total antioxidant capacity improves endothelial function in a low cardiovascular risk population», en Nutr Metab Cardiovasc Dis., 22(1), 2012, pp. 50–57.

[144]. Webb, A. J., Patel, N., Loukogeorgakis, S., et al., «Acute blood pressure lowering, vasoprotective, and antiplatelet properties of dietary nitrate via bioconversion to nitrite», en Hypertension, 51(3), 2008, pp. 784–790

[145]. Smith, R. E., Ashiya, M., «Antihypertensive therapies», en Nat Rev Drug Discov., 6(8), 2007, pp. 597–598.

[146]. Kapil, V., Khambata, R. S., Robertson, A., Caulfield, M. J., Ahluwalia, A., «Dietary nitrate provides sustained blood pressure lowering in hypertensive patients: a randomized, phase 2, doubleblind, placebo-controlled study», en Hypertension, 65(2), 2015, pp. 320–327.

[147]. Wylie, L. J., Kelly, J., Bailey, S. J., et al., «Beetroot juice and exercise: pharmacodynamic and doseresponse relationships», en J Appl Physiol., 115(3), 2013, pp. 325–336.

[148]. European Food Safety Authority, «Nitrate in vegetables: scientific opinion of the panel on contaminants in the food chain», en EFSA J., 689, 2008, pp. 1–79.

[149]. Murphy, M., Eliot, K., Heuertz, R. M., Weiss, E., «Whole beetroot consumption acutely improves running per for mance», en J Acad Nutr Diet., 112(4), 2012, pp. 548–552.

[150]. Clements, W. T., Lee, S. R., Bloomer, R. J., «Nitrate ingestion: a review of the health and physical perfor mance effects», en Nutrients, 6(11), 2014, pp. 5224–5264.

[151]. Hord, N. G., Tang, Y., Bryan, N. S., «Food sources of nitrates and nitrites: the physiologic context for potential health benefits», en Am J Clin Nutr., 90(1), 2009, pp. 1–10.

[152]. Bhupathiraju, S. N., Wedick, N. M., Pan, A., et al., «Quantity and variety in fruit and vegetable intake and risk of coronary heart disease», en Am J Clin Nutr., 98(6), 2013, pp. 1514–1523.

[153]. Tamakoshi, A., Tamakoshi, K., Lin, Y., Yagyu, K., Kikuchi, S., «Healthy lifestyle and preventable death: findings from the Japan Collaborative Cohort ( JACC) Study», en Prev Med., 48(5), 2009, pp. 486–492.

[154]. Wang, F., Dai, S., Wang, M., Morrison, H., «Erectile dysfunction and fruit/vegetable consumption among diabetic Canadian men», en Urology, 82(6), 2013, pp. 1330–1335.

[155]. Presley, T. D., Morgan, A. R., Bechtold, E., et al., «Acute effect of a high nitrate diet on brain perfusion in older adults», en Nitric Oxide, 24(1), 2011, pp. 34–42.

[156]. Engan, H. K., Jones, A. M., Ehrenberg, F., Schagatay, E., «Acute dietary nitrate supplementation improves dry static apnea performance», en Respir Physiol Neurobiol., 182(2–3), 2012, pp. 53–59.

[157]. Bailey, S. J., Winyard, P., Vanhatalo, A., et al., «Dietary nitrate supplementation reduces the O2 cost of low-intensity exercise and enhances tolerance to high-intensity exercise in humans», en J Appl Physiol., 107(4), 2009, pp. 1144–1155.

[158]. Murphy, M., Eliot, K., Heuertz, R. M., Weiss, E., «Whole beetroot consumption acutely improves running performance», en J Acad Nutr Diet, 112(4), 2012, pp. 548–552.

[159]. Lidder, S., Webb, A. J., «Vascular effects of dietary nitrate (as found in green leafy vegetables and beetroot) via the nitrate-nitrite-nitric oxide pathway», en Br J Clin Pharmacol., 75(3), 2013, pp. 677–696.

[160]. Wylie, L. J., Kelly, J., Bailey, S. J., et al., «Beetroot juice and exercise: pharmacodynamic and dose response relationships», en J Appl Physiol., 115(3), 2013, pp. 325–336.

 

 

8. No morir de una enfermedad hepática

 

[1]. Chiras, D. D., Human Biology, Jones & Bartlett Learning, Burlington, Massachussetts, 2015.

[2]. Centers for Disease Control and Prevention, «Deaths: final data for 2013 table 10. Number of deaths from 113 selected causes», en National Vital Statistics Report, 64(2), 2016.

[3]. National Cancer Institute Surveillance, Epidemiology, and End Results Program, «SEER stat fact sheets: liver and intrahepatic bile duct cancer», en <http://seer.cancer.gov/statfacts/html/livibd.html>. Último acceso 3 de mayo de 2015.

[4]. Holubek, W. J., Kalman, S., Hoffman, R. S., «Acetaminophen-induced acute liver failure: results of a United States multicenter, prospective study», en Hepatology, 43(4), 2006, p. 880.

[5]. Mokdad, A. H., Marks, J. S., Stroup, D. F., Gerberding, J. L., «Actual causes of death in the United States, 2000», en JAMA, 291(10), 2004, pp. 1238–1245.

[6]. «CDC Morbidity and Mortality Weekly Report. Alcohol-attributable deaths and years of potential life 1 - United States, 2001», en <http://www.cdc.gov/mmwr/preview/mmwrhtml/mm5337a2.htm>, 24 de septiembre de 2004. Último acceso 2 de marzo de 2015.

[7]. Centers for Disease Control and Prevention, «Fact sheets - alcohol use and your health», en <http://www.cdc.gov/alcohol/fact-sheets/alcohol-use.htm>, 7 de noviembre de 2014. Último acceso 2 de marzo de 2015.

[8]. Schwartz, J. M., Reinus, J. F., «Prevalence and natural history of alcoholic liver disease», en Clin Liver Dis., 16(4), 2012, pp. 659–666.

[9]. Lane, B. P., Lieber, C. S., «Ultrastructural alterations in human hepatocytes following ingestion of ethanol with adequate diets», en Am J Pathol., 49(4), 1966, pp. 593–603.

[10]. Mendenhall, C. L., «Anabolic steroid therapy as an adjunct to diet in alcoholic hepatic steatosis», en Am J Dig Dis., 13(9), 1968, pp. 783–791.

[11]. O’Shea, R. S., Dasarathy, S., McCullough, A. J., «Alcoholic liver disease», en Hepatology, 51(1), 2010, pp. 307–328.

[12]. Mandayam, S., Jamal, M. M., Morgan, T. R., «Epidemiology of alcoholic liver disease», en Semin Liver Dis., 24(3), 2004, pp. 217–232.

[13]. Galambos, J. T., «Natural history of alcoholic hepatitis. 3. Histological changes», en Gastroenterology, 63(6), 1972, pp. 1026–1035.

[14]. Woerle, S., Roeber, J., Landen, M. G., «Prevalence of alcohol dependence among excessive drinkers in New Mexico», en Alcohol Clin Exp Res., 31(2), 2007, pp. 293–298.

[15]. Kaskutas, L. A., «Alcoholics anonymous eff ectiveness: faith meets science», en J Addict Dis., 28(2), 2009, pp. 145–157.

[16]. Grønbaek, M., «The positive and negative health effects of alcohol and the public health implications», en J Intern Med., 265(4), 2009, pp. 407–420.

[17]. Britton, A., Marmot, M. G., Shipley, M., «Who benefits most from the cardioprotective properties of alcohol consumption—health freaks or couch potatoes?», en J Epidemiol Community Health, 62(10), 2008, pp. 905–908.

[18]. Agarwal, D. P., «Cardioprotective effects of light-moderate consumption of alcohol: a review of putative mechanisms», en Alcohol Alcohol., 37(5), 2002, pp. 09–415.

[19]. Britton, A., Marmot, M. G., Shipley, M., «Who benefits most from the cardioprotective properties of alcohol consumption—health freaks or couch potatoes?», en J Epidemiol Community Health, 62(10), 2008, pp. 905–908.

[20]. Britton, A., Marmot, M. G., Shipley, M., «Who benefits most from the cardioprotective properties of alcohol consumption—health freaks or couch potatoes?», en J Epidemiol Community Health, 62(10), 2008, pp. 905–908.

*. Juego de palabras con «Walker», que en inglés significa «caminante». (N. de la t.)

[21]. Kechagias, S., Ernersson, Å., Dahlqvist, O., et al., «Fast-food-based hyper-alimentation can induce rapid and profound elevation of serum alanine aminotransferase in healthy subjects», en Gut, 57(5), 2008, pp. 649–654.

[22]. McCarthy, E. M., Rinella, M. E., «The role of diet and nutrient composition in nonalcoholic fatty liver disease», en J Acad Nutr Diet, 112(3), 2012, pp. 401–409.

[23]. Silverman, J. F., Pories, W. J., Caro, J. F., «Liver pathology in diabetes mellitus and morbid obesity: clinical, pathological and biochemical considerations», en Pathol Annu., 24, 1989, pp. 275–302.

[24]. Singh, S., Allen, A. M., Wang, Z., Prokop, L. J., Murad, M. H., Loomba, R., «Fibrosis progression in nonalcoholic fatty liver vs nonalcoholic steatohepatitis: a systematic review and meta-analysis of paired-biopsy studies», en Clin Gastroenterol Hepatol., S1542–3565(14), 2014, pp. 00602–00608.

[25]. Zelber-Sagi, S., Nitzan-Kaluski, D., Goldsmith, R., et al., «Long term nutritional intake and the risk for non-alcoholic fatty liver disease (NAFLD): a population based study», en J Hepatol., 47(5), 2007, pp. 711–717.

[26]. Zelber-Sagi, S., Nitzan-Kaluski, D., Goldsmith, R., et al., «Long term nutritional intake and the risk for non-alcoholic fatty liver disease (NAFLD): a population based study», en J Hepatol., 47(5), 2007, pp. 711–717.

[27]. Longato, L., «Non-alcoholic fatty liver disease (NAFLD): a tale of fat and sugar?», en Fibrogenesis Tissue Repair, 6(1), 2013, p. 14.

[28]. Musso, G., Gambino, R., De Michieli, F., et al., «Dietary habits and their relations to insulin resistance and postprandial lipemia in nonalcoholic steatohepatitis», en Hepatology, 37(4), 2003, pp. 909–916.

[29]. Kontogianni, M. D., Tileli, N., Margariti, A., et al., «Adherence to the Mediterranean diet is associated with the severity of non-alcoholic fatty liver disease», en Clin Nutr., 33(4), 2014, pp. 678–683.

[30]. Kim, E. J., Kim, B. H., Seo, H. S., et al., «Cholesterol-induced non-alcoholic fatty liver disease and atherosclerosis aggravated by systemic inflammation», en PLoS ONE, 9(6), 2014, p. e97841.

[31]. Yasutake, K., Nakamuta, M., Shima, Y., et al., «Nutritional investigation of non-obese patients with non-alcoholic fatty liver disease: the significance of dietary cholesterol», en Scand J Gastroenterol, 44(4), 2009, pp. 471–477.

[32]. Duewell, P., Kono, H., Rayner, K. J., et al., «NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals that form early in disease», en Nature, 464(7293), 2010, pp. 1357–1361.

[33]. Ioannou, G. N., Haigh, W. G., Thorning, D., Savard, C., «Hepatic cholesterol crystals and crown-like structures distinguish NASH from simple steatosis», en J Lipid Res., 54(5), 2013, pp. 1326–1334.

[34]. U.S. Department of Agriculture Agricultural Research Service, «National Nutrient Database for Standard Reference Release 27. Basic Report: 21359, McDonald’s, sausage McMuffi’n with egg», en <http://ndb.nal.usda.gov/ndb/foods/show/6845>. Último acceso 2 de marzo de 2015.

[35]. Ioannou, G. N., Morrow, O. B., Connole, M. L., Lee, S. P., «Association between dietary nutrient composition and the incidence of cirrhosis or liver cancer in the United States population», en Hepatology, 50(1), 2009, pp. 175–184.

[36]. National Institute of Diabetes and Digestive and Kidney Diseases, «Liver transplantation», en <http://www.niddk.nih.gov/health-information/health-topics/ liver-disease/liver-transplant/Pages/facts.aspx>, junio de 2010. Último acceso 2 de marzo de 2015.

[37]. Kwak, J. H., Baek, S. H., Woo, Y., et al., «Beneficial immunostimulatory effect of short-term Chlorella supplementation: enhancement of natural killer cell activity and early inflammatory response (randomized, double-blinded, placebo-controlled trial)», en Nutr J., 11, 2012, p. 53.

[38]. Azocar, J., Diaz, A., «Efficacy and safety of Chlorella supplementation in adults with chronic hepatitis C virus infection», en World J Gastroenterol., 19(7), 21 de febrero de 2013, pp. 1085–1090.

[39]. Goozner, M., «Why Sovaldi shouldn’t cost $84,000», en Mod Healthc., 44(18), 2014, p. 26.

[40]. Lock, G., Dirscherl, M., Obermeier, F., et al., «Hepatitis C - contamination of toothbrushes: myth or reality?», en J Viral Hepat., 13(9), 2006, pp. 571–573.

[41]. Bocket, L., Chevaliez, S., Talbodec, N., Sobaszek, A., Pawlotsky, J. M., Yazdanpanah, Y., «Occupational transmission of hepatitis C virus resulting from use of the same supermarket meat slicer», en Clin Microbiol Infect., 17(2), 2011, pp. 238–241.

[42]. Teo, C. G., «Much meat, much malady: changing perceptions of the epidemiology of hepatitis E», en Clin Microbiol Infect., 16(1), 2010, pp. 24–32.

[43]. Yazaki, Y., Mizuo, H., Takahashi, M., et al., «Sporadic acute or fulminant hepatitis E in Hokkaido, Japan, may be food-borne, as suggested by the presence of hepatitis E virus in pig liver as food», en J Gen Virol., 84(Pt 9), 2003, pp. 2351–2357.

[44]. Feagins, A. R., Opriessnig, T., Guenette, D. K., Halbur, P. G., Meng, X. J., «Detection and characterization of infectious Hepatitis E virus from commercial pig livers sold in local grocery stores in the USA», en J Gen Virol., 88(Pt 3), 2007, pp. 912–917.

[45]. Feagins, A. R., Opriessnig, T., Guenette, D. K., Halbur, P. G., Meng, X. J., «Detection and characterization of infectious Hepatitis E virus from commercial pig livers sold in local grocery stores in the USA», en J Gen Virol., 88(Pt 3), 2007, pp. 912–917.

[46]. Dalton, H. R., Bendall, R. P., Pritchard, C., Henley, W., Melzer, D., «National mortality rates from chronic liver disease and consumption of alcohol and pig meat», en Epidemiol Infect., 138(2), 2010, pp. 174–182.

[47]. Emerson, S. U., Arankalle, V. A., Purcell, R. H., «Thermal stability of hepatitis E virus», en J Infect Dis., 192(5), 1 de septiembre de 2005, pp. 930–933.

[48]. Centers for Disease Control and Prevention, «What can you do to protect yourself and your family from food poisoning?», en <http://www.cdc.gov/foodsafety/prevention.html>, 6 de septiembre de 2013. Último acceso 11 de marzo de 2015.

[49]. Shinde, N. R., Patil, T. B., Deshpande, A. S., Gulhane, R. V., Patil, M. B., Bansod, Y. V., «Clinical profile, maternal and fetal outcomes of acute hepatitis E in pregnancy», en Ann Med Health Sci Res., 4(Suppl 2), 2014, pp. S133–S139.

[50]. Navarro, V. J., Barnhart, H., Bonkovsky, H. L., et al., «Liver injury from herbals and dietary supplements in the U.S. Drug-Induced Liver Injury Network», en Hepatology, 60(4), 2014, pp. 1399–1408.

[51]. Yu, E. L., Sivagnanam, M., Ellis, L., Huang, J. S., «Acute hepatotoxicity after ingestion of Morinda citrifolia (Noni Berry) juice in a 14-year-old boy», en J Pediatr Gastroenterol Nutr., 52(2), 2011, pp. 222–224.

[52]. Licata, A., Craxt, A., «Considerations regarding the alleged association between Herbalife products and cases of hepatotoxicity: a rebuttal», en Intern Emerg Med., 9(5), 2014, pp. 601–602.

[53]. Lobb, A. L., «Science in liquid dietary supplement promotion: the misleading case of mangosteen juice», en Hawaii J Med Public Health, 71(2), 2012, pp. 46–48.

[54]. Lobb, A. L., «Science in liquid dietary supplement promotion: the misleading case of mangosteen juice», en Hawaii J Med Public Health, 71(2), 2012, pp. 46–48.

[55]. Boozer, C. N., Nasser, J. A., Heymsfield, S. B., Wang, V., Chen, G., Solomon, J. L., «An herbal supplement containing Ma Huang-Guarana for weight loss: a randomized, double-blind trial», en Int J Obes Relat Metab Disord., 25(3), 2001, pp. 316–324.

[56]. US Government Accountability Office, «Dietary Supplements Containing Ephedra: Health Risks and FDA’s Oversight», en <http://www.gao.gov/assets/ 120/110228.pdf>, 23 de julio de 2003. Último acceso 2 de marzo de 2015.

[57]. Preuss, H. G., Bagchi, D., Bagchi, M., Rao, C. V., Dey, D. K., Satyanarayana, S., «Effects of a natural extract of (-)-hydroxycitric acid (HCA-SX) and a combination of HCA-SX plus niacin-bound chromium and Gymnema sylvestre extract on weight loss», en Diabetes Obes Metab., 6(3), 2004, pp. 171–180.

[58]. Fong, T. L., Klontz, K. C., Canas Coto, A., et al., «Hepatotoxicity due to hydroxycut: a case series», en Am J Gastroenterol., 105(7), 2010, pp. 1561– 1566.

[59]. Ye, E. Q., Chacko, S. A., Chou, E. L., Kugizaki, M., Liu, S., «Greater whole-grain intake is associated with lower risk of type 2 diabetes, cardiovascular disease, and weight gain», en J Nutr., 142(7), 2012, pp. 1304–1313.

[60]. Karl, J. P., Saltzman, E., «The role of whole grains in body weight regulation», en Adv Nutr., 3(5), 2012, pp. 697–707.

[61]. Ye, E. Q., Chacko, S. A., Chou, E. L., Kugizaki, M., Liu, S., «Greater whole-grain intake is associated with lower risk of type 2 diabetes, cardiovascular disease, and weight gain», en J Nutr., 142(7), 2012, pp. 1304–1313.

[62]. Chang, H.-C., Huang, C.-N., Yeh, D.-M., Wang, S.-J., Peng, C.-H., Wang, C.-J., «Oat prevents obesity and abdominal fat distribution, and improves liver function in humans», en Plant Foods Hum Nutr., 68(1), 2013, pp. 18–23.

[63]. Chang, H.-C., Huang, C.-N., Yeh, D.-M., Wang, S.-J., Peng, C.-H., Wang, C.-J., «Oat prevents obesity and abdominal fat distribution, and improves liver function in humans», en Plant Foods Hum Nutr., 68(1), 2013, pp. 18–23.

[64]. Georgoulis, M., Kontogianni, M. D., Tileli, N., et al., «The impact of cereal grain consumption on the development and severity of non-alcoholic fatty liver disease», en Eur J Nutr., 53(8), 2014, pp. 1727–1735.

[65]. Valenti, L., Riso, P., Mazzocchi, A., Porrini, M., Fargion, S., Agostoni, C., «Dietary anthocyanins as nutritional therapy for nonalcoholic fatty liver disease», en Oxid Med Cell Longev., 2013, 2013, p. 145421.

[66]. Suda, I., Ishikawa, F., Hatakeyama, M., et al., «Intake of purple sweet potato beverage affects on serum hepatic biomarker levels of healthy adult men with borderline hepatitis», en Eur J Clin Nutr., 62(1), 2008, pp. 60–67.

[67]. Sun, J., Chu, Y. F., Wu, X., Liu, R. H., «Antioxidant and antiproliferative activities of common fruits», en J Agric Food Chem., 50(25), 2002, pp. 7449–7454.

[68]. Ferguson, P. J., Kurowska, E. M., Freeman, D. J., Chambers, A. F., Koropatnick, J., «In vivo inhibition of growth of human tumor lines by flavonoid fractions from cranberry extract», en Nutr Cancer, 56(1), 2006, pp. 86–94.

[69]. Sun, J., Hai Liu, R., «Cranberry phytochemical extracts induce cell cycle arrest and apoptosis in human MCF-7 breast cancer cells», en Cancer Lett., 241(1), 2006, pp. 124–134.

[70]. Ferguson, P. J., Kurowska, E. M., Freeman, D. J., Chambers, A. F., Koropatnick, J., «In vivo inhibition of growth of human tumor lines by flavonoid fractions from cranberry extract», en Nutr Cancer, 56(1), 2006, pp. 86–94.

[71]. Kresty, L. A., Howell, A. B., Baird, M., «Cranberry proanthocyanidins mediate growth arrest of lung cancer cells through modulation of gene expression and rapid induction of apoptosis», en Molecules, 16(3), 2011, pp. 2375–2390.

[72]. Seeram, N. P., Adams, L. S., Zhang, Y., et al., «Blackberry, black raspberry, blueberry, cranberry, red raspberry, and strawberry extracts inhibit growth and stimulate apoptosis of human cancer cells in vitro», en J Agric Food Chem., 54(25), 2006, pp. 9329–9339.

[73]. Kim, K. K., Singh, A. P., Singh, R. K., et al., «Anti-angiogenic activity of cranberry proanthocyanidins and cytotoxic properties in ovarian cancer cells», en Int J Oncol., 40(1), 2012, pp. 227–235.

[74]. Déziel, B., MacPhee, J., Patel, K., et al., «American cranberry (Vaccinium macrocarpon) extract affects human prostate cancer cell growth via cell cycle arrest by modulating expression of cell cycle regulators», en Food Funct., 3(5), 2012, pp. 556–564.

[75]. Liu, M., Lin, L. Q., Song, B. B., et al., «Cranberry phytochemical extract inhibits SGC-7901 cell growth and human tumor xenografts in Balb/c nu/nu mice», en J Agric Food Chem., 57(2), 2009, pp. 762–768.

[76]. Seeram, N. P., Adams, L. S., Hardy, M. L., Heber, D., «Total cranberry extract versus its phytochemical constituents: antiproliferative and synergistic effects against human tumor cell lines», en J Agric Food Chem., 52(9), 2004, pp. 2512–2517.

[77]. Grace, M. H., Massey, A. R., Mbeunkui, F., Yousef, G. G., Lila, M. A., «Comparison of health-relevant flavonoids in commonly consumed cranberry products», en J Food Sci., 77(8), 2012, pp. H176–H183.

[78]. Grace, M. H., Massey, A. R., Mbeunkui, F., Yousef, G. G., Lila, M. A., «Comparison of health-relevant flavonoids in commonly consumed cranberry products», en J Food Sci., 77(8), 2012, pp. H176–H183.

[79]. Vinson, J. A., Bose, P., Proch, J., Al Kharrat, H., Samman, N., «Cranberries and cranberry products: powerful in vitro, ex vivo, and in vivo sources of antioxidants», en J Agric Food Chem., 56(14), 2008, pp. 5884–5891.

[80]. White, B. L., Howard, L. R., Prior, R. L., «Impact of different stages of juice processing on the anthocyanin, flavonol, and procyanidin contents of cranberries», en J Agric Food Chem., 59(9), 2011, pp. 4692–4698.

[81]. Arnesen, E., Huseby, N.-E., Brenn, T., Try, K., «The Tromse heart study: distribution of, and determinants for, gamma-glutamyltransferase in a free-living population», en Scand J Clin Lab Invest., 46(1), 1986, pp. 63–70.

[82]. Ruhl, C. E., Everhart, J. E., «Coffee and tea consumption are associated with a lower incidence of chronic liver disease in the United States», en Gastroenterology, 129(6), 2005, pp. 1928–1936.

[83]. Salgia, R., Singal, A. G., «Hepatocellular carcinoma and other liver lesions», en Med Clin North Am., 98(1), 2014, pp. 103–118.

[84]. Sang, L. X., Chang, B., Li, X.-H., Jiang, M., «Consumption of coff ee associated with reduced risk of liver cancer: a meta-analysis», en BMC Gastroenterol., 13, 2013, p. 34.

[85]. Lai, G. Y., Weinstein, S. J., Albanes, D., et al., «The association of coff ee intake with liver cancer incidence and chronic liver disease mortality in male smokers», en Br J Cancer, 109(5), 2013, pp. 1344–1351.

[86]. Fujita, Y., Shibata, A., Ogimoto, I., et al., «The effect of interaction between hepatitis C virus and cigarette smoking on the risk of hepatocellular carcinoma», en Br J Cancer, 94(5), 2006, pp. 737–739.

[87]. Danielsson, J., Kangastupa, P., Laatikainen, T., Aalto, M., Niemelä, O., «Dose- and gender-dependent interactions between coffee consumption and serum GGT activity in alcohol consumers», en Alcohol Alcohol., 48(3), 2013, pp. 303–307.

[88]. Bravi, F., Bosetti, C., Tavani, A., Gallus, S., La Vecchia, C., «Coffee reduces risk for hepatocellular carcinoma: an meta-analysis», en Clin Gastroenterol Hepatol., 11(11), 2013, pp. 1413–1421.e1.

[89]. Browning, J. D., Szczepaniak, L. S., Dobbins, R., et al., «Prevalence of hepatic steatosis in an urban population in the United States: impact of ethnicity», en Hepatology, 40(6), 2004, pp. 1387–1395.

[90]. Cardin, R., Piciocchi, M., Martines, D., Scribano, L., Petracco, M., Farinati, F., «Effects of coffee consumption in chronic hepatitis C: a randomized controlled trial», en Dig Liver Dis., 45(6), 2013, pp. 499–504.

[91]. Torres, D. M., Harrison, S. A., «Is it time to write a prescription for coffee? Coffee and liver disease», en Gastroenterology, 144(4), 2013, pp. 670–672.

[92]. Ng, V., Saab, S., «Can daily coffee consumption reduce liver disease-related mortality?», en Clin Gastroenterol Hepatol., 11(11), 2013, pp. 1422–1423.

[93]. Torres, D. M., Harrison, S. A., «Is it time to write a prescription for coffee? Coffee and liver disease», en Gastroenterology, 144(4), 2013, pp. 670–672.

[94]. Juliano, L. M., Griffiths, R. R., A critical review of caffeine withdrawal: empirical validation of symptoms and signs, incidence, severity, and associated features», en Psychopharmacology (Berlin), 176(1), 2004, pp. 1–29.

[95]. O’Keefe, J. H., Bhatti, S. K., Patil, H. R., DiNicolantonio, J. J., Lucan, S. C., Lavie, C. J., «Effects of habitual coffee consumption on cardiometabolic disease, cardiovascular health, and all-cause mortality», en J Am Coll Cardiol., 62(12), 2013, pp. 1043–1051.

 

 

9. No morir de un cáncer en la sangre

 

[1]. Hunger, S. P., Lu, X., Devidas, M., et al., «Improved survival for children and adolescents with acute lymphoblastic leukemia between 1990 and 2005: a report from the children’s oncology group», en J Clin Oncol., 30(14), 2012, pp. 1663–1669.

[2]. National Cancer Institute Surveillance, Epidemiology, and End Results Program, «SEER Stat Fact Sheets: Leukemia», en <http://seer.cancer.gov/statfacts/html/leuks.html>. Último acceso 15 de junio de 2015.

[3]. «American Cancer Society, Cancer Facts & Figures 2014», American Cancer Society, Atlanta, 2014.

[4]. «American Cancer Society, Cancer Facts & Figures 2014», American Cancer Society, Atlanta, 2014.

[5]. «American Cancer Society, Cancer Facts & Figures 2014», American Cancer Society, Atlanta, 2014.

[6]. Key, T. J., Appleby, P. N., Spencer, E. A., et al., «Cancer incidence in British vegetarians», en Br J Cancer, 101(1), 2009, pp. 192–197.

[7]. «Vegetarians less likely to develop cancer than meat eaters» (nota de prensa), en British Journal of Cancer, <http://www.nature.com/bjc/press_releases/p_r_jul09_6605098.html>, Londres, UK, 1 de julio de 2009. Último acceso 11 de marzo de 2015.

[8]. Suppipat, K., Park, C. S., Shen, Y., Zhu, X., Lacorazza, H. D., «Sulforaphane induces cell cycle arrest and apoptosis in acute lymphoblastic leukemia cells», en PLoS One, 7(12), 2012, p. e51251.

[9]. Han, X., Zheng, T., Foss, F., et al., «Vegetable and fruit intake and non-Hodgkin lymphoma survival in Connecticut women», en Leuk Lymphoma, 51(6), 2010, pp. 1047–1054.

[10]. Thompson, C. A., Cerhan, J. R., «Fruit and vegetable intake and survival from non-Hodgkin lymphoma: does an apple a day keep the doctor away?», en Leuk Lymphoma, 51(6), 2010, pp. 963–964.

[11]. Thompson, C. A., Habermann, T. M., Wang, A. H., et al., «Antioxidant intake from fruits, vegetables and other sources and risk of non-Hodgkin’s lymphoma: the Iowa Women’s Health Study», en Int J Cancer, 126(4), 2010, pp. 992–1003.

[12]. Holtan, S. G., O’Connor, H. M., Fredericksen, Z. S., et al., «Food-frequency questionnaire-based estimates of total antioxidant capacity and risk of non-Hodgkin lymphoma», en Int J Cancer, 131(5), 2012, pp. 1158–1168.

[13]. Holtan, S. G., O’Connor, H. M., Fredericksen, Z. S., et al., «Food-frequency questionnaire-based estimates of total antioxidant capacity and risk of non-Hodgkin lymphoma», en Int J Cancer, 131(5), 2012, pp. 1158–1168.

[14]. Thompson, C. A., Habermann, T. M., Wang, A. H., et al., «Antioxidant intake from fruits, vegetables and other sources and risk of non-Hodgkin’s lymphoma: the Iowa Women’s Health Study», en Int J Cancer, 126(4), 2010, pp. 992–1003.

[15]. Bjelakovic, G., Nikolova, D., Simonetti, R. G., Gluud, C., «Antioxidant supplements for prevention of gastrointestinal cancers: a systematic review and meta-analysis», en Lancet, 364(9441), 2004, pp. 1219–1228.

[16]. Jacobs, D. R., Tapsell, L. C., «Food synergy: the key to a healthy diet», en Proc Nutr Soc., 72(2), 2013, pp. 200–206.

[17]. Elsayed, R. K., Glisson, J. K., Minor, D. S., «Rhabdomyolysis associated with the use of a mislabeled “acai berry” dietary supplement», en Am J Med Sci., 342(6), 2011, pp. 535–538.

[18]. Zhang, Y., Wang, D., Lee, R. P., Henning, S. M., Heber, D., «Absence of pomegranate ellagitannins in the majority of commercial pomegranate extracts: implications for standardization and quality control», en J Agric Food Chem., 57(16), 2009, pp. 7395–7400.

[19]. Zhang, Y., Krueger, D., Durst, R., et al., «International multidimensional authenticity specifi cation (IMAS) algorithm for detection of commercial pomegranate juice adulteration», en J Agric Food Chem., 57(6), 2009, pp. 2550–2557.

[20]. Del Pozo-Insfran, D., Percival, S. S., Talcott, S. T., «Açai (Euterpe oleracea Mart.) polyphenolics in their glycoside and aglycone forms induce apoptosis of HL-60 leukemia cells», en J Agric Food Chem., 54(4), 2006, pp. 1222–1229.

[21]. Schauss, A. G., Wu, X., Prior, R. L., et al., «Antioxidant capacity and other bioactivities of the freezedried Amazonian palm berry, Euterpe oleraceae mart. (aSch)», en J Agric Food Chem., 54(22), 2006, pp. 8604–8610.

[22]. Jensen, G. S., Ager, D. M., Redman, K. A., Mitzner, M. A., Benson, K. F., Schauss, A. G., «Pain reduction and improvement in range of motion after daily consumption of an açai (Euterpe oleracea Mart.) pulp-fortified polyphenolic-rich fruit and berry juice blend», en J Med Food, 14(7–8), 2011, pp. 702–711.

[23]. Udani, J. K., Singh, B. B., Singh, V. J., Barrett, M. L., «Effects of açai (Euterpe oleracea Mart.) berry preparation on metabolic parameters in a healthy overweight population: a pilot study», en Nutr J., 10, 2011, p. 45.

[24]. Haytowitz, D. B., Bhagwat, S. A., «USDA database for the oxygen radical capacity (ORAC) of selected foods, release 2», United States Department of Agriculture, Washington, D. C., 2010.

[25]. «American Cancer Society, Cancer Facts & Figures 2014», American Cancer Society, Atlanta, 2014.

[26]. Landgren, O., Kyle, R. A., Pfeiffer, R. M., et al., «Monoclonal gammopathy of undetermined significance (MGUS) consistently precedes multiple myeloma: a prospective study», en Blood, 113(22), 2009, pp. 5412–5417.

[27]. Landgren, O., Kyle, R. A., Pfeiffer, R. M., et al., «Monoclonal gammopathy of undetermined significance (MGUS) consistently precedes multiple myeloma: a prospective study», en Blood, 113(22), 2009, pp. 5412–5417.

[28]. Greenberg, A. J., Vachon, C. M., Rajkumar, S. V., «Disparities in the prevalence, pathogenesis and progression of monoclonal gammopathy of undetermined signifi cance and multiple myeloma between blacks and whites», en Leukemia, 26(4), 2012, pp. 609–614.

[29]. Kyle, R. A., Therneau, T. M., Rajkumar, S. V., et al., «A long-term study of prognosis in monoclonal gammopathy of undetermined significance», en N Engl J Med., 346(8), 2002, pp. 564–569.

[30]. Bharti, A. C., Donato, N., Singh, S., Aggarwal, B. B., «Curcumin (diferuloylmethane) down-regulates the constitutive activation of nuclear factor-kappa B and IkappaBalpha kinase in human multiple myeloma cells, leading to suppression of proliferation and induction of apoptosis», en Blood, 101(3), 2003, pp. 1053–1062.

[31]. Golombick, T., Diamond, T. H., Badmaev, V., Manoharan, A., Ramakrishna, R., «The potential role of curcumin in patients with monoclonal gammopathy of undefined significance—its effect on paraproteinemia and the urinary N-telopeptide of type I collagen bone turnover marker», en Clin Cancer Res., 15(18), 2009, pp. 5917–5922.

[32]. Golombick, T., Diamond, T. H., Manoharan, A., Ramakrishna, R., «Monoclonal gammopathy of undetermined significance, smoldering multiple myeloma, and curcumin: a randomized, doubleblind placebo-controlled cross-over 4g study and an open-label 8g extension study», en Am J Hematol., 87(5), 2012, pp. 455–460.

[33]. Key, T. J., Appleby, P. N., Spencer, E. A., et al., «Cancer incidence in British vegetarians», en Br J Cancer, 101(1), 2009, pp. 192–197.

[34]. Rohrmann, S., Linseisen, J., Jakobsen, M. U., et al., «Consumption of meat and dairy and lymphoma risk in the European Prospective Investigation into Cancer and Nutrition», en Int J Cancer, 128(3), 2011, pp. 623–634.

[35]. U.S. Department of Agriculture Agricultural Research Service, «National Nutrient Database for Standard Reference Release 27. Basic Report: 05358, chicken, broiler, rotisserie, BBQ, breast meat and skin», en <http://ndb.nal.usda.gov/ndb/foods/show/1058>. Último acceso 2 de marzo de 2015.

[36]. Rohrmann, S., Linseisen, J., Jakobsen, M. U., et al., «Consumption of meat and dairy and lymphoma risk in the European Prospective Investigation into Cancer and Nutrition», en Int J Cancer, 128(3), 2011, pp. 623–634.

[37]. Chiu, B. C., Cerhan, J. R., Folsom, A. R., et al., «Diet and risk of non-Hodgkin lymphoma in older women», en JAMA, 275(17), 1996, pp. 1315–1321.

[38]. Daniel, C. R., Sinha, R., Park, Y., et al., «Meat intake is not associated with risk of non-Hodgkin lymphoma in a large prospective cohort of U.S. men and women», en J Nutr., 142(6), 2012, pp. 1074–1080.

[39]. Puangsombat, K., Gadgil, P., Houser, T. A., Hunt, M. C., Smith, J. S., «Occurrence of heterocyclic amines in cooked meat products», en Meat Sci., 90(3), 2012, pp. 739–746.

[40]. ’T Mannetje, A., Eng, A., Pearce, N., «Farming, growing up on a farm, and haematological cancer mortality», en Occup Environ Med., 69(2), 2012, pp. 126–132.

[41]. Johnson, E. S., Zhou, Y., Yau, L. C., et al., «Mortality from malignant diseases-update of the Baltimore union poultry cohort», en Cancer Causes Control, 21(2), 2010, pp. 215–221.

[42]. Neasham, D., Sifi, A., Nielsen, K. R., et al., «Occupation and risk of lymphoma: a multicenter prospective cohort study (EPIC)», en Occup Environ Med., 68(1), 2011, pp. 77–81.

[43]. Kalland, K.H., Ke, X.S., Øyan, A. M., «Tumour virology—history, status and future challenges», en APMIS, 117(5–6), 2009, pp. 382–399.

[44]. Centers for Disease Control and Prevention, «Human Orf virus infection from house hold exposures—United States, 2009–2011», en MMWR Morb Mortal Wkly Rep., 61(14), 2012, pp. 245–248.

[45]. Benton, E. C., «Warts in butchers—a cause for concern?», en Lancet, 343(8906), 1994, p. 1114.

[46]. Gubéran Usel, M., Raymond, L., Fioretta, G., «Mortality and incidence of cancer among a cohort of self employed butchers from Geneva and their wives», en Br J Ind Med., 50(11), 1993, pp. 1008–1016.

[47]. Johnson, E. S., Zhou, Y., Yau, L. C., et al., «Mortality from malignant diseases-update of the Baltimore union poultry cohort», en Cancer Causes Control, 21(2), 2010, pp. 215–221.

[48]. Johnson, E. S., Ndetan, H., Lo, K. M., «Cancer mortality in poultry slaughtering/processing plant workers belonging to a union pension fund», en Environ Res., 110(6), 2010, pp. 588–594.

[49]. Choi, K. M., Johnson, E. S., «Occupational exposure assessment using antibody levels: exposure to avian leukosis/sarcoma viruses in the poultry industry», en Int J Environ Health Res., 21(4), 2011, pp. 306–316.

[50]. Choi, K. M., Johnson, E. S., «Industrial hygiene assessment of reticuloendotheliosis viruses exposure in the poultry industry», en Int Arch Occup Environ Health, 84(4), 2011, pp. 375–382.

[51]. Choi, K. M., Johnson, E. S., «Industrial hygiene assessment of reticuloendotheliosis viruses exposure in the poultry industry», en Int Arch Occup Environ Health, 84(4), 2011, pp. 375–382.

[52]. Johnson, E. S., Ndetan, H., Lo, K. M., «Cancer mortality in poultry slaughtering/processing plant workers belonging to a union pension fund», en Environ Res., 110(6), 2010, pp. 588–594.

[53]. ’T Mannetje, A., Eng, A., Pearce, N., «Farming, growing up on a farm, and haematological cancer mortality», en Occup Environ Med., 69(2), 2012, pp. 126–132.

[54]. Tranah, G. J., Bracci, P. M., Holly, E. A., «Domestic and farm-animal exposures and risk of non-Hodgkin’s lymphoma in a population-based study in the San Francisco Bay Area», en Cancer Epidemiol Biomarkers Prev., 17(9), 2008, pp. 2382–2387.

[55]. Buehring, G. C., Philpott, S. M., Choi, K. Y., «Humans have antibodies reactive with Bovine leukemia virus», en AIDS Res Hum Retroviruses, 19(12), 2003, pp. 1105–1113.

[56]. U.S. Department of Agriculture Animal and Plant Health Inspection Service, «Bovine Leukosis Virus (BLV) on U.S. Dairy Operations, 2007», en <http://www.aphis.usda.gov/animal_health/nahms/dairy/downloads/dairy07/Dairy07_is_BLV.pdf>, octubre de 2008. Último acceso 2 de marzo de 2015.

[57]. Buehring, G. C., Shen, H. M., Jensen, H. M., Choi, K. Y., Sun, D., Nuovo, G., «Bovine leukemia virus DNA in human breast tissue», en Emerging Infect Dis., 20(5), 2014, pp. 772–782.

[58]. Tranah, G. J., Bracci, P. M., Holly, E. A., «Domestic and farm-animal exposures and risk of non-Hodgkin’s lymphoma in a population-based study in the San Francisco Bay Area», en Cancer Epidemiol Biomarkers Prev., 17(9), 2008, pp. 2382–2387.

[59]. Schernhammer, E. S., Bertrand, K. A., Birmann, B. M., Sampson, L., Willett, W. C., Feskanich, D., «Consumption of artificial sweetener- and sugar-containing soda and risk of lymphoma and leukemia in men and women», en Am J Clin Nutr., 96(6), 2012, pp. 1419–1428.

[60]. Lim, U., Subar, A. F., Mouw, T., et al., «Consumption of aspartame-containing beverages and incidence of hematopoietic and brain malignancies», en Cancer Epidemiol Biomarkers Prev., 15(9), 2006, pp. 1654–1659.

[61]. McCullough, M. L., Teras, L. R., Shah, R., Diver, W. R., Gaudet, M. M., Gapstur, S. M., Artificially and sugar-sweetened carbonated beverage consumption is not associated with risk of lymphoid neoplasms in older men and women», en J Nutr., 144(12), 2014, pp. 2041–2049.

 

 

10. No morir de insuficiencia renal

 

[1]. Stokes, J. B., «Consequences of frequent hemodialysis: comparison to conventional hemodialysis and transplantation», en Trans Am Clin Climatol Assoc., 122, 2011, pp. 124–36.

[2]. Coresh, J., Selvin, E., Stevens, L. A., et al., «Prevalence of chronic kidney disease in the United States», en JAMA, 298(17), 2007, pp. 2038–2047.

[3]. Stevens, L. A., Li, S., Wang, C., et al., «Prevalence of CKD and comorbid illness in el derly patients in the United States: results from the Kidney Early Evaluation Program (KEEP)», en Am J Kidney Dis., 55(3 Suppl 2), 2010, pp. S23–S33.

[4]. Ryan, T. P., Sloand, J. A., Winters, P. C., Corsetti, J. P., Fisher, S. G., «Chronic kidney disease prevalence and rate of diagnosis», en Am J Med., 120(11), 2007, pp. 981–986.

[5]. Hoerger, T. J., Simpson, S. A., Yarnoff, B. O., et al., «The future burden of CKD in the United States: a simulation model for the CDC CKD Initiative», en Am J Kidney Dis., 65(3), 2015, pp. 403–411.

[6]. Dalrymple, L. S., Katz, R., Kestenbaum, B., et al., «Chronic kidney disease and the risk of end-stage renal disease versus death», en J Gen Intern Med., 26(4), 2011, pp. 379–385.

[7]. Kumar, S., Bogle, R., Banerjee, D., «Why do young people with chronic kidney disease die early?», en World J Nephrol., 3(4), 2014, pp. 143–155.

[8]. Lin, J., Hu, F. B., Curhan, G. C., «Associations of diet with albuminuria and kidney function decline», en Clin J Am Soc Nephrol., 5(5), 2010, pp. 836–843.

[9]. Lin, J., Hu, F. B., Curhan, G. C., «Associations of diet with albuminuria and kidney function decline», en Clin J Am Soc Nephrol., 5(5), 2010, pp. 836–843.

[10]. Virchow, R., Cellular Pathology as Based upon Physiological and Pathological Histology. Twenty Lectures Delivered in the Pathological Institute of Berlin During the Months of February, March and April, 1858, J. B. Lippincott and Co., Filadelfia, PA, 1863.

[11]. Moorhead, J. F., Chan, M. K., El-Nahas, M., Varghese, Z., «Lipid nephrotoxicity in chronic progressive glomerular and tubulo-interstitial disease», en Lancet, 2(8311), 1982, pp. 1309–1311.

[12]. Hartroft, W. S., «Fat emboli in glomerular capillaries of choline-deficient rats and of patients with diabetic glomerulosclerosis», en Am J Pathol., 31(3), 1955, pp. 381–397.

[13]. Gyebi, L., Soltani, Z., Reisin, E., «Lipid nephrotoxicity: new concept for an old disease. Curr Hypertens Rep., 14(2), 2012, pp. 177–181.

[14]. US Burden of Disease Collaborators, «The state of US health, 1990–2010: burden of diseases, injuries, and risk factors», en JAMA, 310(6), 14 de agosto de 2013, pp. 591–608.

[15]. Odermatt, A., «The Western-style diet: a major risk factor for impaired kidney function and chronic kidney disease», en Am J Physiol Renal Physiol., 301(5), 2011, pp. F919–F931.

[16]. Van den Berg, E., Hospers, F. A., Navis, G., et al., «Dietary acid load and rapid progression to endstage renal disease of diabetic nephropathy in Westernized South Asian people», en J Nephrol., 24(1), 2011, pp. 11–17.

[17]. Piccoli, G. B., Vigotti, F. N., Leone, F., et al., «Low-protein diets in CKD: how can we achieve them? A narrative, pragmatic review», en Clin Kidney J., 8(1), 2015, pp. 61–70.

[18]. Brenner, B. M., Meyer, T. W., Hostetter, T. H., «Dietary protein intake and the progressive nature of kidney disease: the role of hemodynamically mediated glomerular injury in the pathogenesis of progressive glomerular sclerosis in aging, renal ablation, and intrinsic renal disease», en N Engl J Med., 307(11), 9 de septiembre de 1982, pp. 652–659.

[19]. Wiseman, M. J., Hunt, R., Goodwin, A., Gross, J. L., Keen, H., Viberti, G. C., «Dietary composition and renal function in healthy subjects», en Nephron, 46(1), 1987, pp. 37–42.

[20]. Nakamura, H., Takasawa, M., Kashara, S., et al., «Effects of acute protein loads of different sources on renal function of patients with diabetic nephropathy», en Tohoku J Exp Med., 159(2), 1989, pp. 153–162.

[21]. Simon, A. H., Lima, P. R., Almerinda, M., Alves, V. F., Bottini, P. V., De Faria, J. B., «Renal haemodynamic responses to a chicken or beef meal in normal individuals», en Nephrol Dial Transplant, 13(9), 1998, pp. 2261–2264.

[22]. Kontessis, P., Jones, S., Dodds, R., et al., «Renal, metabolic and hormonal responses to ingestion of animal and vegetable proteins», en Kidney Int., 38(1), 1990, pp. 136–144.

[23]. Nakamura, H., Takasawa, M., Kashara, S., et al., «Effects of acute protein loads of different sources on renal function of patients with diabetic nephropathy», en Tohoku J Exp Med., 159(2), 1989, pp. 153–162.

[24]. Azadbakht, L., Shakerhosseini, R., Atabak, S., Jamshidian, M., Mehrabi, Y., Esmaill-Zadeh, A., «Beneficiary effect of dietary soy protein on lowering plasma levels of lipid and improving kidney function in type II diabetes with nephropathy», en Eur J Clin Nutr., 57(10), 2003, pp. 1292–1294.

[25]. Kontessis, P. A., Bossinakou, I., Sarika, L., et al., «Renal, metabolic, and hormonal responses to proteins of different origin in normotensive, nonproteinuric type I diabetic patients», en Diabetes Care, 18(9), 1995, pp. 1233–1240.

[26]. Teixeira, S. R., Tappenden, K. A., Carson, L., et al., «Isolated soy protein consumption reduces urinary albumin excretion and improves the serum lipid profile in men with type 2 diabetes mellitus and nephropathy», en J Nutr., 134(8), 2004, pp. 1874–1880.

[27]. Stephenson, T. J., Setchell, K. D., Kendall, C. W., Jenkins, D. J., Anderson, J. W., Fanti, P., «Effect of soy protein-rich diet on renal function in young adults with insulin-dependent diabetes mellitus», en Clin Nephrol., 64(1), 2005, pp. 1–11.

[28]. Jibani, M. M., Bloodworth, L. L., Foden, E., Griffiths, K. D., Galpin, O. P., «Predominantly vegetarian diet in patients with incipient and early clinical diabetic nephropathy: effects on albumin excretion rate and nutritional status», en Diabet Med., 8(10), 1991, pp. 949–953.

[29]. Bosch, J. P., Saccaggi, A., Lauer, A., Ronco, C., Belledonne, M., Glabman, S., «Renal functional reserve in humans. Effect of protein intake on glomerular filtration rate», en Am J Med., 75(6), 1983, pp. 943–950.

[30]. Liu, Z. M., Ho, S. C., Chen, Y. M., Tang, N., Woo, J., «Effect of whole soy and purified isoflavone daidzein on renal function—a 6-month randomized controlled trial in equol-producing postmenopausal women with prehypertension», en Clin Biochem., 47(13–14), 2014, pp. 1250–1256.

[31]. Fioretto, P., Trevisan, R., Valerio, A., et al., «Impaired renal response to a meat meal in insulindependent diabetes: role of glucagon and prostaglandins», en Am J Physiol., 258(3 Pt 2), 1990, pp. F675–F683.

[32]. Frassetto, L., Morris, R. C., Sellmeyer, D. E., Todd, K., Sebastian, A., «Diet, evolution and aging—the pathophysiologic effects of the post-agricultural inversion of the potassium-to-sodium and baseto chloride ratios in the human diet», en Eur J Nutr., 40(5), 2001, pp. 200–213.

[33]. Banerjee, T., Crews, D. C., Wesson, D. E., et al. «Dietary acid load and chronic kidney disease among adults in the United States», en BMC Nephrol., 15, 24 de agosto de 2014, p. 137.

[34]. Sebastian, A., Frassetto, L. A., Sellmeyer, D. E., Merriam, R. L., Morris, R. C., «Estimation of the net acid load of the diet of ancestral preagricultural Homo sapiens and their hominid ancestors», en Am J Clin Nutr., 76(6), 2002, pp. 1308–1316.

[35]. Van den Berg, E., Hospers, F. A., Navis, G., et al., «Dietary acid load and rapid progression to endstage renal disease of diabetic nephropathy in Westernized South Asian people», en J Nephrol., 24(1), 2011, pp. 11–17.

[36]. Uribarri, J., Oh, M. S., «The key to halting progression of CKD might be in the produce market, not in the pharmacy», en Kidney Int., 81(1), 2012, pp. 7–9.

[37]. Cohen, E., Nardi, Y., Krause, I., et al., «A longitudinal assessment of the natural rate of decline in renal function with age», en J Nephrol., 27(6), 2014, pp. 635–641.

[38]. Brenner, B. M., Meyer, T. W., Hostetter, T. H., «Dietary protein intake and the progressive nature of kidney disease: the role of hemodynamically mediated glomerular injury in the pathogenesis of progressive glomerular sclerosis in aging, renal ablation, and intrinsic renal disease», en N Engl J Med., 307(11), 9 de septiembre de 1982, pp. 652–659.

[39]. Frassetto, L. A., Todd, K. M., Morris, R. C., Sebastian, A., «Estimation of net endogenous noncarbonic acid production in humans from diet potassium and protein contents», en Am J Clin Nutr., 68(3), 1998, pp. 576–583.

[40]. Wiseman, M. J., Hunt, R., Goodwin, A., Gross, J. L., Keen, H., Viberti, G. C., «Dietary composition and renal function in healthy subjects», en Nephron, 46(1), 1987, pp. 37–42.

[41]. Kempner, W., «Treatment of heart and kidney disease and of hypertensive and arteriosclerotic vascular disease with the rice diet», en Ann Intern Med., 31(5), 1949, pp. 821–856.

[42]. Barsotti, G., Morelli, E., Cupisti, A., Meola, M., Dani, L., Giovannetti, S., «A low-nitrogen lowphosphorus vegan diet for patients with chronic renal failure», en Nephron, 74(2), 1996, pp. 390–394.

[43]. Deriemaeker, P., Aerenhouts, D., Hebbelinck, M., Clarys, P., «Nutrient based estimation of acid-base balance in vegetarians and non-vegetarians», en Plant Foods Hum Nutr., 65(1), 2010, pp. 77–82.

[44]. Goraya, N., Simoni, J., Jo, C., Wesson, D. E., «Dietary acid reduction with fruits and vegetables or bicarbonate attenuates kidney injury in patients with a moderately reduced glomerular filtration rate due to hypertensive nephropathy», en Kidney Int., 81(1), 2012, pp. 86–93.

[45]. Yaqoob, M. M., «Treatment of acidosis in CKD», en Clin J Am Soc Nephrol., 8(3), 2013, pp. 342–343.

[46]. Goraya, N., Simoni, J., Jo, C., Wesson, D. E., «Dietary acid reduction with fruits and vegetables or bicarbonate attenuates kidney injury in patients with a moderately reduced glomerular filtration rate due to hypertensive nephropathy», en Kidney Int., 81(1), 2012, pp. 86–93.

[47]. Wright, J. A., Cavanaugh, K. L., «Dietary sodium in chronic kidney disease: a comprehensive approach», en Semin Dial., 23(4), 2010, pp. 415–421.

[48]. Uribarri, J., Oh, M. S., «The key to halting progression of CKD might be in the produce market, not in the pharmacy», en Kidney Int., 81(1), 2012, pp. 7–9.

[49]. Goldfarb, S., «Dietary factors in the pathogenesis and prophylaxis of calcium nephrolithiasis», en Kidney Int., 34(4), 1988, pp. 544–555.

[50]. Scales, C. D. Jr., Smith, A. C., Hanley, J. M., Saigal, C. S., «Urologic Diseases in America Project. Prevalence of kidney stones in the United States», en Eur Urol., 62(1), 2012, pp. 160–165.

[51]. Robertson, W. G., Peacock, M., Hodgkinson, A., «Dietary changes and the incidence of urinary calculi in the U.K. between 1958 and 1976», en J Chronic Dis., 32(6), 1979, pp. 469–476.

[52]. Robertson, W. G., Heyburn, P. J., Peacock, M., Hanes, F. A., Swaminathan, R., «The effect of high animal protein intake on the risk of calcium stone-formation in the urinary tract», en Clin Sci (Londres)., 57(3), 1979, pp. 285–288.

[53]. Robertson, W. G., Heyburn, P. J., Peacock, M., Hanes, F. A., Swaminathan, R., «The effect of high animal protein intake on the risk of calcium stone-formation in the urinary tract», en Clin Sci (Londres)., 57(3), 1979, pp. 285–288.

[54]. Robertson, W. G., Peacock, M., Heyburn, P. J., et al., «Should recurrent calcium oxalate stone formers become vegetarians?», en Br J Urol., 51(6), 1979, pp. 427–431.

[55]. Turney, B. W., Appleby, P. N., Reynard, J. M., Noble, J. G., Key, T. J., Allen, N. E., «Diet and risk of kidney stones in the Oxford cohort of the European Prospective Investigation into Cancer and Nutrition (EPIC)», en Eur J Epidemiol., 29(5), 2014, pp. 363–369.

[56]. Tracy, C. R., Best, S., Bagrodia, A., et al., «Animal protein and the risk of kidney stones: A comparative metabolic study of animal protein sources», en J Urol., 192, 8 de febrero de 2014, pp. 137–141.

[57]. Bushinsky, D. A., «Recurrent hypercalciuric nephrolithiasis—does diet help?», en N Engl J Med., 346(2), 10 de enero de 2002, pp. 124–125.

[58]. Borghi, L., Schianchi, T., Meschi, T., et al., «Comparison of two diets for the prevention of recurrent stones in idiopathic hypercalciuria», en N Engl J Med., 346(2), 10 de enero de 2002, pp. 77–84.

[59]. Sorensen, M. D., Hsi, R. S., Chi, T., et al., «Dietary intake of fi ber, fruit and vegetables decreases the risk of incident kidney stones in women: a Women’s Health Initiative report», en J Urol., 192(6), 2014, pp. 1694–1699.

[60]. Mehta, T. H., Goldfarb, D. S., «Uric acid stones and hyperuricosuria», en Adv Chronic Kidney Dis., 19(6), 2012, pp. 413–418.

[61]. De Vries, A., Frank, M., Liberman, U. A., Sperling, O., «Allopurinol in the prophylaxis of uric acid stones», en Ann Rheum Dis., 25(6 Suppl), 1966, pp. 691–693.

[62]. Siener, R., Hesse, A., «The effect of a vegetarian and different omnivorous diets on urinary risk factors for uric acid stone formation», en Eur J Nutr., 42(6), 2003, pp. 332–337.

[63]. Siener, R., Hesse, A., «The effect of a vegetarian and different omnivorous diets on urinary risk factors for uric acid stone formation», en Eur J Nutr., 42(6), 2003, pp. 332–337.

*. Carga ácida renal potencial. (N. de la t.)

[64]. Trinchieri, A., «Development of a rapid food screener to assess the potential renal acid load of diet in renal stone formers (LAKE score)», en Arch Ital Urol Androl., 84(1), 2012, pp. 36–38.

[65]. Chae, J. Y., Kim, J. W., Kim, J. W., et al., «Increased fluid intake and adequate dietary modification may be enough for the successful treatment of uric acid stone», en Urolithiasis, 41(2), 2013, pp. 179–182.

[66]. Deriemaeker, P., Aerenhouts, D., Hebbelinck, M., Clarys, P., «Nutrient based estimation of acid-base balance in vegetarians and non-vegetarians», en Plant Foods Hum Nutr., 65(1), 2010, pp. 77–82.

[67]. Adeva, M. M., Souto, G., «Diet-induced metabolic acidosis», en Clin Nutr., 30(4), 2011, pp. 416–421.

[68]. Dawson-Hughes, B., Harris, S. S., Ceglia, L., «Alkaline diets favor lean tissue mass in older adults», en Am J Clin Nutr., 87(3), 2008, pp. 662–665.

[69]. Ritz, E., Hahn, K., Ketteler, M., Kuhlmann, M. K., Mann, J., «Phosphate additives in food—a health risk», en Dtsch Arztebl Int., 109(4), 2012, pp 49–55.

[70]. Ritz, E., Hahn, K., Ketteler, M., Kuhlmann, M. K., Mann, J., «Phosphate additives in food—a health risk», en Dtsch Arztebl Int., 109(4), 2012, pp. 49–55.

[71]. Calvo, M. S., Uribarri, J., «Public health impact of dietary phosphorus excess on bone and cardiovascular health in the general population», en Am J Clin Nutr., 98(1), 2013, pp. 6–15.

[72]. Moe, S. M., Zidehsarai, M. P., Chambers, M. A., et al., «Vegetarian compared with meat dietary protein source and phosphorus homeostasis in chronic kidney disease», en Clin J Am Soc Nephrol., 6(2), 2011, pp. 257–264.

[73]. Fukagawa, M., Komaba, H., Miyamoto, K., «Source matters: from phosphorus load to bioavailability», en Clin J Am Soc Nephrol., 6(2), 2011, pp. 239–240.

[74]. Murphy-Gutekunst, L., Uribarri, J., «Hidden phosphorus-enhanced meats: Part 3», en J Ren Nutr., 15(4), 2005, pp. E1–E4.

[75]. Ritz, E., Hahn, K., Ketteler, M., Kuhlmann, M. K., Mann, J., «Phosphate additives in food—a health risk», en Dtsch Arztebl Int., 109(4), 2012, pp. 49–55.

[76]. Karp, H., Ekholm, P., Kemi, V., et al., «Differences among total and in vitro digestible phosphorus content of plant foods and beverages», en J Ren Nutr., 22(4), 2012, pp. 416–422.

[77]. Karp, H., Ekholm, P., Kemi, V., Hirvonen, T., Lamberg-Allardt, C., «Differences among total and in vitro digestible phosphorus content of meat and milk products», en J Ren Nutr., 22(3), 2012, pp. 344–349.

[78]. Karp, H., Ekholm, P., Kemi, V., et al., «Differences among total and in vitro digestible phosphorus content of plant foods and beverages», en J Ren Nutr., 22(4), 2012, pp. 416–422.

[79]. Murphy-Gutekunst, L., Uribarri, J., «Hidden phosphorus-enhanced meats: Part 3», en J Ren Nutr., 15(4), 2005, pp. E1–E4.

[80]. Sherman, R. A., Mehta, O., «Phosphorus and potassium content of enhanced meat and poultry products: implications for patients who receive dialysis», en Clin J Am Soc Nephrol., 4(8), 2009, pp. 1370–1373.

[81]. Benini, O., D’Alessandro, C., Gianfaldoni, D., Cupisti, A., «Extra-phosphate load from food additives in commonly eaten foods: a real and insidious danger for renal patients», en J Ren Nutr., 21(4), 2011, pp. 303–308.

[82]. Sherman, R. A., Mehta, O., «Phosphorus and potassium content of enhanced meat and poultry products: implications for patients who receive dialysis», en Clin J Am Soc Nephrol., 4(8), 2009, pp. 1370–1373.

[83]. Benini, O., D’Alessandro, C., Gianfaldoni, D., Cupisti, A., «Extra-phosphate load from food additives in commonly eaten foods: a real and insidious danger for renal patients», en J Ren Nutr., 21(4), 2011, pp. 303–308.

[84]. Shroff, R., «Phosphate is a vascular toxin», en Pediatr Nephrol., 28(4), 2013, pp. 583–593.

[85]. Shuto, E., Taketani, Y., Tanaka, R., et al., «Dietary phosphorus acutely impairs endothelial function», en J Am Soc Nephrol., 20(7), 2009, pp. 1504–1512.

[86]. Gunther, N. W., He, Y., Fratamico, P., «Effects of polyphosphate additives on the pH of processed chicken exudates and the survival of Campylobacter», en J Food Prot., 74(10), 2011, pp. 1735–1740.

[87]. Sherman, R. A., Mehta, O., «Dietary phosphorus restriction in dialysis patients: potential impact of processed meat, poultry, and fish products as protein sources», en Am J Kidney Dis., 54(1), 2009, pp. 18–23.

[88]. Sherman, R. A., Mehta, O., «Dietary phosphorus restriction in dialysis patients: potential impact of processed meat, poultry, and fish products as protein sources», en Am J Kidney Dis., 54(1), 2009, pp. 18–23.

[89]. Sullivan, C. M., Leon, J. B., Sehgal, A. R., «Phosphorus-containing food additives and the accuracy of nutrient databases: implications for renal patients», en J Ren Nutr., 17(5), 2007, pp. 350–354.

[90]. Food and Drug Administration, Department of Health and Human Services, «Final Determination Regarding Partially Hydrogenated Oils. Docket No. FDA-2013-N-1317», en <https://s3.amazonaws.com/public-inspection.federalregister.gov/2015-14883.pdf>, 16 de junio de 2015. Último acceso 16 de junio de 2015.

[91]. Food and Drug Administration, Department of Health and Human Services, «Tentative determination regarding partially hydrogenated oils; request for comments and for scientific data and information. Federal Register Docket No. D78 FR 67169-75», en <https://www.federalregister.gov/articles/2013/11/08/2013-2685 4/tentative-determination-regarding-partially-hydrogenated-oils-request-for-comments-and-for>, 8 de noviembre de 2013. Último acceso 2 de marzo de 2015.

[92]. Food and Drug Administration, Department of Health and Human Services, «Tentative determination regarding partially hydrogenated oils; request for comments and for scientific data and information. Federal Register Docket No. D78 FR 67169-75», en <https://www.federalregister.gov/articles/2013/11/08/2013-2685 4/tentative-determination-regarding-partially-hydrogenated-oils-request-for-comments-and-for>, 8 de noviembre de 2013. Último acceso 2 de marzo de 2015.

[93]. Neltner, T. G., Kulkami, N. R., Alger, H. M., et al., «Navigating the U.S. food additive regulatory program», en Compr Rev Food Sci Food Saf., 10(6), 2011, pp. 342–368.

[94]. Neltner, T. G., Alger, H. M., O’Reilly, J. T., Krimsky, S., Bero, L. A., Maffini, M. V., «Conflicts of interest in approvals of additives to food determined to be generally recognized as safe: out of balance», en JAMA Intern Med., 173(22), 2013, pp. 2032–2036.

[95]. Stuckler, D., Basu, S., McKee, M., «Commentary: UN high level meeting on non-communicable diseases: an opportunity for whom?», en BMJ, 343, 2011, p. d5336.

[96]. Moodie, R., Stuckler, D., Monteiro, C., et al., «Profits and pandemics: prevention of harmful effects of tobacco, alcohol, and ultra-processed food and drink industries», en Lancet, 381(9867), 2013, pp. 670–679.

[97]. American Cancer Society, «Cancer Facts & Figures 2014», American Cancer Society, Atlanta, 2014.

[98]. Kirkali, Z., Cal, C., «Renal Cell Carcinoma: Overview», en Nargund, V. H., Raghavan, D., Sandler, H. M. (comps.), Urological Oncology, Springer, Londres, 2008, pp. 263–280.

[99]. American Cancer Society, «Cancer Facts & Figures 2014», American Cancer Society, Atlanta, 2014.

[100]. Ramírez, N., Özel, M. Z., Lewis, A. C., Marcé, R. M., Borrull, F., Hamilton, J. F., «Exposure to nitrosamines in thirdhand tobacco smoke increases cancer risk in non-smokers», en Environ Int., 71, 2014, pp. 139–147.

[101]. Schick, S. F., Farraro, K. F., Perrino, C., et al., «Thirdhand cigarette smoke in an experimental chamber: evidence of surface deposition of nicotine, nitrosamines and polycyclic aromatic hydrocarbons and de novo formation of NNK», en Tob Control, 23(2), 2014, pp. 152–159.

[102]. Hecht, S. S., «It is time to regulate carcinogenic tobacco-speciffic nitrosamines in cigarette tobacco», en Cancer Prev Res. (Filadelfia), 7(7), 2014, pp. 639–647.

[103]. Rodgman, A., Perfetti, T. A., The Chemical Components of Tobacco and Tobacco Smoke, CRC Press, Taylor & Francis Group, Boca Ratón, Florida, 2009.

[104]. Haorah, J., Zhou, L., Wang, X., Xu, G., Mirvish, S. S., «Determination of total N-nitroso compounds and their precursors in frankfurters, fresh meat, dried salted fish, sauces, tobacco, and tobacco smoke particulates», en J Agric Food Chem., 49(12), 2001, pp. 6068–6078.

[105]. Rohrmann, S., Overvad, K., Bueno de Mesquita, H. B., et al., «Meat consumption and mortality—results from the European Prospective Investigation into Cancer and Nutrition», en BMC Med., 11, 2013, p. 63.

[106]. Sinha, R., Cross, A. J., Graubard, B. I., Leitzmann, M. F., Schatzkin, A., «Meat intake and mortality: a prospective study of over half a million people», en Arch Intern Med., 169(6), 2009, pp. 562–571.

[107]. American Institute for Cancer Research, «Recommendations for Cancer Prevention», en <http://www.aicr.org/reduce-your-cancer-risk/recommendations-for-cancer-prevention/recommendations_05_red_meat.html>, 17 de abril de 2011. Último acceso 2 de marzo de 2015.

[108]. USDA, «Additives in meat and poultry products», en <http://www.fsis.usda.gov/wps/portal/fsis/topics/food-safety-education/get-answers/food-safety-fact-sheets/food-labeling/additives-in-meat-andpoultry-products/additives-in-meatand-poultry-products>, 24 de marzo de 2015. Último acceso 3 de mayo de 2015.

[109]. Sebranek, J. G., Jackson-Davis, A. L., Myers, K. L., Lavieri, N. A., «Beyond celery and starter culture: advances in natural/organic curing processes in the United States», en Meat Sci., 92(3), 2012, pp. 267–273.

[110]. Dellavalle, C. T., Daniel, C. R., Aschebrook-Kilfoy, B., et al., «Dietary intake of nitrate and nitrite and risk of renal cell carcinoma in the NIH-AARP Diet and Health Study», en Br J Cancer, 108(1), 2013, pp. 205–212.

[111]. Bartsch, H., Ohshima, H., Pignatelli, B., «Inhibitors of endogenous nitrosation. Mechanisms and implications in human cancer prevention», en Mutat Res., 202(2), 1988, pp. 307–324.

[112]. Dellavalle, C. T., Daniel, C. R., Aschebrook-Kilfoy, B., et al., «Dietary intake of nitrate and nitrite and risk of renal cell carcinoma in the NIH-AARP Diet and Health Study», en Br J Cancer, 108(1), 2013, pp. 205–212.

[113]. Liu, B., Mao, Q., Wang, X., et al., «Cruciferous vegetables consumption and risk of renal cell carcinoma: a meta-analysis», en Nutr Cancer, 65(5), 2013, pp. 668–676.